The Nuclear Receptor AhR Controls Bone Homeostasis by Regulating Osteoclast Differentiation via the RANK/c-Fos Signaling Axis
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Title
The Nuclear Receptor AhR Controls Bone Homeostasis by Regulating Osteoclast Differentiation via the RANK/c-Fos Signaling Axis
Authors
Keywords
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Journal
JOURNAL OF IMMUNOLOGY
Volume 197, Issue 12, Pages 4639-4650
Publisher
The American Association of Immunologists
Online
2016-11-15
DOI
10.4049/jimmunol.1600822
References
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Note: Only part of the references are listed.- Alternative NF-κB Regulates RANKL-Induced Osteoclast Differentiation and Mitochondrial Biogenesis via Independent Mechanisms
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- PGC1β Mediates PPARγ Activation of Osteoclastogenesis and Rosiglitazone-Induced Bone Loss
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- Hypersensitivity of Aryl Hydrocarbon Receptor-Deficient Mice to Lipopolysaccharide-Induced Septic Shock
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- Coordination of PGC-1β and iron uptake in mitochondrial biogenesis and osteoclast activation
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- Inducibility of cytochrome P450 1A1 and chemical carcinogenesis by benzo[a]pyrene in AhR repressor-deficient mice
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