MicroRNA-106b is involved in transforming growth factor β1–induced cell migration by targeting disabled homolog 2 in cervical carcinoma
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Title
MicroRNA-106b is involved in transforming growth factor β1–induced cell migration by targeting disabled homolog 2 in cervical carcinoma
Authors
Keywords
MicroRNA-106b, Cervical cancer, Transforming growth factor β1, Disabled homolog 2
Journal
JOURNAL OF EXPERIMENTAL & CLINICAL CANCER RESEARCH
Volume 35, Issue 1, Pages -
Publisher
Springer Nature
Online
2016-01-15
DOI
10.1186/s13046-016-0290-6
References
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Note: Only part of the references are listed.- Remodeling of extracellular matrix by normal and tumor-associated fibroblasts promotes cervical cancer progression
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- The microRNA-218~Survivin axis regulates migration, invasion, and lymph node metastasis in cervical cancer
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- Down-regulation of miRNA-106b inhibits growth of melanoma cells by promoting G1-phase cell cycle arrest and reactivation of p21/WAF1/Cip1 protein
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- Disabled-2 downregulation promotes epithelial-to-mesenchymal transition
- (2010) J C Martin et al. BRITISH JOURNAL OF CANCER
- Epigenetic downregulation of human disabled homolog 2 switches TGF-β from a tumor suppressor to a tumor promoter
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- HPV16 E7 oncogene expression in normal human epithelial cells causes molecular changes indicative of an epithelial to mesenchymal transition
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- Molecular Diagnosis of Sentinel Lymph Node Metastases in Cervical Cancer Using Squamous Cell Carcinoma Antigen
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- MicroRNA-134 Modulates the Differentiation of Mouse Embryonic Stem Cells, Where It Causes Post-Transcriptional Attenuation of Nanog and LRH1
- (2007) Yvonne M.-S. Tay et al. STEM CELLS
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