TRAP5b and RANKL/OPG Predict Bone Pathology in Patients with Gaucher Disease
Published 2021 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
TRAP5b and RANKL/OPG Predict Bone Pathology in Patients with Gaucher Disease
Authors
Keywords
-
Journal
Journal of Clinical Medicine
Volume 10, Issue 10, Pages 2217
Publisher
MDPI AG
Online
2021-05-20
DOI
10.3390/jcm10102217
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Effect of Substrate Reduction Therapy in Comparison to Enzyme Replacement Therapy on Immune Aspects and Bone Involvement in Gaucher Disease
- (2020) Renuka P. Limgala et al. Biomolecules
- Cathepsin K: The Action in and Beyond Bone
- (2020) Rongchen Dai et al. Frontiers in Cell and Developmental Biology
- Value of Glucosylsphingosine (Lyso-Gb1) as a Biomarker in Gaucher Disease: A Systematic Literature Review
- (2020) Shoshana Revel-Vilk et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- Local Production of Osteoprotegerin by Osteoblasts Suppresses Bone Resorption
- (2020) Keisha M. Cawley et al. Cell Reports
- Gaucher Disease in Bone: From Pathophysiology to Practice
- (2019) Derralynn Hughes et al. JOURNAL OF BONE AND MINERAL RESEARCH
- Osteoimmunology of Bone Loss in Inflammatory Rheumatic Diseases
- (2019) Fabienne Coury et al. Frontiers in Immunology
- Gaucheromas: When macrophages promote tumor formation and dissemination
- (2018) Margarita Ivanova et al. BLOOD CELLS MOLECULES AND DISEASES
- Common signalling pathways in macrophage and osteoclast multinucleation
- (2018) Marie Pereira et al. JOURNAL OF CELL SCIENCE
- Impact of sphingolipids on osteoblast and osteoclast activity in Gaucher disease
- (2018) Matthew C. Reed et al. MOLECULAR GENETICS AND METABOLISM
- Gene variants of osteoprotegerin, estrogen-, calcitonin- and vitamin D-receptor genes and serum markers of bone metabolism in patients with Gaucher disease type 1
- (2018) Anca Zimmermann et al. Therapeutics and Clinical Risk Management
- Glucosylsphingosine Causes Hematological and Visceral Changes in Mice—Evidence for a Pathophysiological Role in Gaucher Disease
- (2017) Jan Lukas et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- Chronic pain in Gaucher disease: skeletal or neuropathic origin?
- (2017) Grazia Devigili et al. Orphanet Journal of Rare Diseases
- Clonal Immunoglobulin against Lysolipids in the Origin of Myeloma
- (2016) Shiny Nair et al. NEW ENGLAND JOURNAL OF MEDICINE
- Proinflammatory and proosteoclastogenic potential of peripheral blood mononuclear cells from Gaucher patients: Implication for bone pathology
- (2015) J.M. Mucci et al. BLOOD CELLS MOLECULES AND DISEASES
- Macrophages: Their Emerging Roles in Bone
- (2015) Benjamin P Sinder et al. JOURNAL OF BONE AND MINERAL RESEARCH
- Pathogenesis of Bone Alterations in Gaucher Disease: The Role of Immune System
- (2015) Juan Marcos Mucci et al. Journal of Immunology Research
- Management of Bone Disease in Gaucher Disease Type 1: Clinical Practice
- (2014) Gaetano Giuffrida et al. ADVANCES IN THERAPY
- Type II NKT-TFH cells against Gaucher lipids regulate B-cell immunity and inflammation
- (2014) S. Nair et al. BLOOD
- Tartrate-resistant acid phosphatase 5b is a marker of osteoclast number and volume in RAW 264.7 cells treated with receptor-activated nuclear κB ligand
- (2014) YUANDONG LV et al. Experimental and Therapeutic Medicine
- Biology of the RANKL–RANK–OPG System in Immunity, Bone, and Beyond
- (2014) Matthew C. Walsh et al. Frontiers in Immunology
- Enhanced differentiation of osteoclasts from mononuclear precursors in patients with Gaucher disease
- (2013) M. Reed et al. BLOOD CELLS MOLECULES AND DISEASES
- Effect of monocytes/macrophages on the early osteogenic differentiation of hBMSCs
- (2012) R. P. Pirraco et al. Journal of Tissue Engineering and Regenerative Medicine
- Inflammatory bone loss: pathogenesis and therapeutic intervention
- (2012) Kurt Redlich et al. NATURE REVIEWS DRUG DISCOVERY
- Managing Macrophages in Rheumatoid Arthritis by Reform or Removal
- (2012) Jun Li et al. Current Rheumatology Reports
- Effect of glycosphingolipids on osteoclastogenesis and osteolytic bone diseases
- (2012) Adel Ersek et al. Frontiers in Endocrinology
- Elevated plasma glucosylsphingosine in Gaucher disease: relation to phenotype, storage cell markers, and therapeutic response
- (2011) N. Dekker et al. BLOOD
- The chemokine CCL18 causes maturation of cultured monocytes to macrophages in the M2 spectrum
- (2011) Ingrid U. Schraufstatter et al. IMMUNOLOGY
- Applications and performance of monoclonal antibodies to human tartrate resistant acid phosphatase
- (2011) Silvia D. Potenziani Pradella et al. JOURNAL OF IMMUNOLOGICAL METHODS
- Therapeutic approaches to bone pathology in Gaucher disease: Past, present and future
- (2011) Ozlem Goker-Alpan MOLECULAR GENETICS AND METABOLISM
- Glucocerebrosidase gene-deficient mouse recapitulates Gaucher disease displaying cellular and molecular dysregulation beyond the macrophage
- (2010) P. K. Mistry et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Bone events and evolution of biologic markers in Gaucher disease before and during treatment
- (2010) Jerome Stirnemann et al. ARTHRITIS RESEARCH & THERAPY
- Control of RANKL gene expression
- (2009) Charles A. O'Brien BONE
- Functions of RANKL/RANK/OPG in bone modeling and remodeling
- (2008) Brendan F. Boyce et al. ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS
- The role of Dickkopf-1 in bone development, homeostasis, and disease
- (2008) J. J. Pinzone et al. BLOOD
- Management of non-neuronopathic Gaucher disease with special reference to pregnancy, splenectomy, bisphosphonate therapy, use of biomarkers and bone disease monitoring
- (2008) T. M. Cox et al. JOURNAL OF INHERITED METABOLIC DISEASE
Find the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
SearchCreate your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create Now