- Home
- Publications
- Publication Search
- Publication Details
Title
Cardiac effects and clinical applications of MG53
Authors
Keywords
-
Journal
Cell and Bioscience
Volume 11, Issue 1, Pages -
Publisher
Springer Science and Business Media LLC
Online
2021-06-28
DOI
10.1186/s13578-021-00629-x
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- MG53 is not a critical regulator of insulin signaling pathway in skeletal muscle
- (2021) Clothilde Philouze et al. PLoS One
- MG53 does not manifest the development of diabetes in db/db mice
- (2020) Qiang Wang et al. DIABETES
- Cardiac Ischemic Preconditioning Promotes MG53 Secretion Through H 2 O 2 -Activated PKC-δ Signaling
- (2020) Dan Shan et al. CIRCULATION
- Glucose-sensitive Myokine/Cardiokine MG53 Regulates Systemic Insulin Response and Metabolic Homeostasis
- (2019) Hong-Kun Wu et al. CIRCULATION
- MG53 Protein Protects Aortic Valve Interstitial Cells From Membrane Injury and Fibrocalcific Remodeling
- (2019) T. M. Ayodele Adesanya et al. Journal of the American Heart Association
- TRIM72 contributes to cardiac fibrosis via regulating STAT3/Notch-1 signaling
- (2019) Xu Chen et al. JOURNAL OF CELLULAR PHYSIOLOGY
- MG53, a Novel Regulator of KChIP2 and I to,f, Plays a Critical Role in Electrophysiological Remodeling in Cardiac Hypertrophy
- (2019) Wenjuan Liu et al. CIRCULATION
- Sustained elevation of MG53 in the bloodstream increases tissue regenerative capacity without compromising metabolic function
- (2019) Zehua Bian et al. Nature Communications
- Implication of altered ubiquitin-proteasome system and ER stress in the muscle atrophy of diabetic rats
- (2018) S. Sreenivasa Reddy et al. ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS
- Potential role of MG53 in the regulation of transforming-growth-factor-β1-induced atrial fibrosis and vulnerability to atrial fibrillation
- (2018) Jingwen Guo et al. EXPERIMENTAL CELL RESEARCH
- MG53 is dispensable for T-tubule maturation but critical for maintaining T-tubule integrity following cardiac stress
- (2017) Caimei Zhang et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Hypercholesterolemia Abrogates Remote Ischemic Preconditioning-Induced Cardioprotection
- (2017) Lei-Lei Ma et al. SHOCK
- Tripartite Motif Protein 72 Regulates the Proliferation and Migration of Rat Cardiac Fibroblasts via the Transforming Growth Factor-β Signaling Pathway
- (2016) Jianquan Zhao et al. CARDIOLOGY
- Lack of MG53 in human heart precludes utility as a biomarker of myocardial injury or endogenous cardioprotective factor
- (2016) Frances A. Lemckert et al. CARDIOVASCULAR RESEARCH
- The Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3)
- (2016) Mervyn Singer et al. JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION
- Swimming Exercise Alleviated Insulin Resistance by Regulating Tripartite Motif Family Protein 72 Expression and AKT Signal Pathway in Sprague-Dawley Rats Fed with High-Fat Diet
- (2016) Jie Qi et al. Journal of Diabetes Research
- TRIF promotes angiotensin II-induced cross-talk between fibroblasts and macrophages in atrial fibrosis
- (2015) Xiao-Qing Chen et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Upregulation of MG53 Induces Diabetic Cardiomyopathy Through Transcriptional Activation of Peroxisome Proliferation-Activated Receptor
- (2015) F. Liu et al. CIRCULATION
- Altered Skeletal Muscle Mitochondrial Proteome As the Basis of Disruption of Mitochondrial Function in Diabetic Mice
- (2015) Piotr Zabielski et al. DIABETES
- Modulation of Wound Healing and Scar Formation by MG53 Protein-mediated Cell Membrane Repair
- (2015) Haichang Li et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Zinc Binding to MG53 Protein Facilitates Repair of Injury to Cell Membranes
- (2015) Chuanxi Cai et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Cardioprotection of recombinant human MG53 protein in a porcine model of ischemia and reperfusion injury
- (2015) Jianxun Liu et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Effect of Metabolic Syndrome on Mitsugumin 53 Expression and Function
- (2015) Hanley Ma et al. PLoS One
- Proteomic profiling of the extracellular matrix (slime sheath) ofDictyostelium discoideum
- (2015) Robert J. Huber et al. PROTEOMICS
- MG53-mediated cell membrane repair protects against acute kidney injury
- (2015) Pu Duann et al. Science Translational Medicine
- Proteomic mapping of proteins released during necrosis and apoptosis from cultured neonatal cardiac myocytes
- (2014) Kurt D. Marshall et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Increased Interaction With Insulin Receptor Substrate 1, a Novel Abnormality in Insulin Resistance and Type 2 Diabetes
- (2014) Michael Caruso et al. DIABETES
- S-nitrosylation of TRIM72 at cysteine 144 is critical for protection against oxidation-induced protein degradation and cell death
- (2014) Mark J. Kohr et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Effect of recombinant human MG53 protein on tourniquet-induced ischemia-reperfusion injury in rat muscle
- (2014) Benjamin T. Corona et al. MUSCLE & NERVE
- Compensation of the AKT signaling by ERK signaling in transgenic mice hearts overexpressing TRIM72
- (2013) Young-Mi Ham et al. EXPERIMENTAL CELL RESEARCH
- Hypercholesterolemia blocked sevoflurane-induced cardioprotection against ischemia–reperfusion injury by alteration of the MG53/RISK/GSK3β signaling
- (2013) Lei-Lei Ma et al. INTERNATIONAL JOURNAL OF CARDIOLOGY
- Central role of E3 ubiquitin ligase MG53 in insulin resistance and metabolic disorders
- (2013) Ruisheng Song et al. NATURE
- Proteomic Analysis of Skeletal Muscle in Insulin-Resistant Mice: Response to 6-Week Aerobic Exercise
- (2013) Hairui Yuan et al. PLoS One
- Hypercholesterolemic Myocardium Is Vulnerable to Ischemia-Reperfusion Injury and Refractory to Sevoflurane-Induced Protection
- (2013) Yong Xu et al. PLoS One
- MG53-induced IRS-1 ubiquitination negatively regulates skeletal myogenesis and insulin signalling
- (2013) Jae-Sung Yi et al. Nature Communications
- Enhancing Muscle Membrane Repair by Gene Delivery of MG53 Ameliorates Muscular Dystrophy and Heart Failure in δ-Sarcoglycan-deficient Hamsters
- (2012) Bo He et al. MOLECULAR THERAPY
- Recombinant MG53 Protein Modulates Therapeutic Cell Membrane Repair in Treatment of Muscular Dystrophy
- (2012) N. Weisleder et al. Science Translational Medicine
- MG53 participates in ischaemic postconditioning through the RISK signalling pathway
- (2011) Yan Zhang et al. CARDIOVASCULAR RESEARCH
- Cardiac Postconditioning
- (2010) Pasquale Pagliaro et al. ANTIOXIDANTS & REDOX SIGNALING
- Mitsugumin 53-mediated maintenance of K+currents in cardiac myocytes
- (2010) Haruko Masumiya et al. Channels
- MG53 Constitutes a Primary Determinant of Cardiac Ischemic Preconditioning
- (2010) Chun-Mei Cao et al. CIRCULATION
- Cardioprotection of Ischemia/Reperfusion Injury by Cholesterol-Dependent MG53-Mediated Membrane Repair
- (2010) Xianhua Wang et al. CIRCULATION RESEARCH
- Ischaemic postconditioning protects against reperfusion injury via the SAFE pathway
- (2009) Lydia Lacerda et al. CARDIOVASCULAR RESEARCH
- No RISK, no ... cardioprotection? A critical perspective
- (2009) G. Heusch CARDIOVASCULAR RESEARCH
- Membrane Repair Defects in Muscular Dystrophy Are Linked to Altered Interaction between MG53, Caveolin-3, and Dysferlin
- (2009) Chuanxi Cai et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Activation of the protective Survivor Activating Factor Enhancement (SAFE) pathway against reperfusion injury: Does it go beyond the RISK pathway?
- (2009) Sandrine Lecour JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Membrane repair redux: redox of MG53
- (2009) Paul McNeil NATURE CELL BIOLOGY
- MG53 Regulates Membrane Budding and Exocytosis in Muscle Cells
- (2008) Chuanxi Cai et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- MG53 nucleates assembly of cell membrane repair machinery
- (2008) Chuanxi Cai et al. NATURE CELL BIOLOGY
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started