Postnatal knockdown ofdok-7gene expression in mice causes structural defects in neuromuscular synapses and myasthenic pathology
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Title
Postnatal knockdown ofdok-7gene expression in mice causes structural defects in neuromuscular synapses and myasthenic pathology
Authors
Keywords
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Journal
GENES TO CELLS
Volume 21, Issue 6, Pages 670-676
Publisher
Wiley
Online
2016-04-19
DOI
10.1111/gtc.12370
References
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- Pharmacologic Treatment of Downstream of Tyrosine Kinase 7 Congenital Myasthenic Syndrome
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- (2012) A. R. Amenta et al. JOURNAL OF NEUROSCIENCE
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- (2012) Melanie A. Samuel et al. PLoS One
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- (2011) Annemart Koornneef et al. MOLECULAR THERAPY
- Dok-7 regulates neuromuscular synapse formation by recruiting Crk and Crk-L
- (2010) P. T. Hallock et al. GENES & DEVELOPMENT
- Dok-7 Activates the Muscle Receptor Kinase MuSK and Shapes Synapse Formation
- (2009) A. Inoue et al. Science Signaling
- Dok-7 myasthenia: Phenotypic and molecular genetic studies in 16 patients
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- Lrp4 Is a Receptor for Agrin and Forms a Complex with MuSK
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- (2008) F. Chevessier et al. HUMAN MOLECULAR GENETICS
- LRP4 Serves as a Coreceptor of Agrin
- (2008) Bin Zhang et al. NEURON
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