The chloride channel CFTR is not required for cyst growth in an ADPKD mouse model
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Title
The chloride channel CFTR is not required for cyst growth in an ADPKD mouse model
Authors
Keywords
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Journal
FASEB JOURNAL
Volume 35, Issue 10, Pages -
Publisher
Wiley
Online
2021-09-02
DOI
10.1096/fj.202100843r
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- Cyst growth in ADPKD is prevented by pharmacological and genetic inhibition of TMEM16A in vivo
- (2020) Ines Cabrita et al. Nature Communications
- Plasma membrane–localized TMEM16 proteins are indispensable for expression of CFTR
- (2019) Roberta Benedetto et al. JOURNAL OF MOLECULAR MEDICINE-JMM
- Pharmacological reversal of renal cysts from secretion to absorption suggests a potential therapeutic strategy for managing autosomal dominant polycystic kidney disease
- (2019) Murali K. Yanda et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Divergent function of polycystin 1 and polycystin 2 in cell size regulation
- (2019) Amandine Viau et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Compartmentalized crosstalk of CFTR and TMEM16A (ANO1) through EPAC1 and ADCY1
- (2018) Joana Lérias et al. CELLULAR SIGNALLING
- HIF-1α promotes cyst progression in a mouse model of autosomal dominant polycystic kidney disease
- (2018) Andre Kraus et al. KIDNEY INTERNATIONAL
- Epithelial Chloride Transport by CFTR Requires TMEM16A
- (2017) Roberta Benedetto et al. Scientific Reports
- P2Y2R is a direct target of HIF-1α and mediates secretion-dependent cyst growth of renal cyst-forming epithelial cells
- (2016) Andre Kraus et al. Purinergic Signalling
- Hypoxia-Inducible Factor-1 Causes Renal Cyst Expansion through Calcium-Activated Chloride Secretion
- (2013) B. Buchholz et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
- Anoctamin 1 induces calcium-activated chloride secretion and proliferation of renal cyst–forming epithelial cells
- (2013) Bjoern Buchholz et al. KIDNEY INTERNATIONAL
- TMEM16A Induces MAPK and Contributes Directly to Tumorigenesis and Cancer Progression
- (2012) U. Duvvuri et al. CANCER RESEARCH
- DOG1: a novel marker of salivary acinar and intercalated duct differentiation
- (2012) Jacinthe Chênevert et al. MODERN PATHOLOGY
- Enhanced Expression of ANO1 in Head and Neck Squamous Cell Carcinoma Causes Cell Migration and Correlates with Poor Prognosis
- (2012) Christian Ruiz et al. PLoS One
- Fluid transport and cystogenesis in autosomal dominant polycystic kidney disease
- (2011) Sara Terryn et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE
- Formation of cysts by principal-like MDCK cells depends on the synergy of cAMP- and ATP-mediated fluid secretion
- (2011) Bjoern Buchholz et al. JOURNAL OF MOLECULAR MEDICINE-JMM
- Why kidneys fail in autosomal dominant polycystic kidney disease
- (2011) Jared J. Grantham et al. Nature Reviews Nephrology
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- (2010) Rainer Schreiber et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- ATP release from non-excitable cells
- (2009) Helle A. Praetorius et al. Purinergic Signalling
- Small-Molecule CFTR Inhibitors Slow Cyst Growth in Polycystic Kidney Disease
- (2008) B. Yang et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
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