Temporal and Spatial Dynamics of Inflammasome Activation After Ischemic Stroke
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Title
Temporal and Spatial Dynamics of Inflammasome Activation After Ischemic Stroke
Authors
Keywords
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Journal
Frontiers in Neurology
Volume 12, Issue -, Pages -
Publisher
Frontiers Media SA
Online
2021-04-24
DOI
10.3389/fneur.2021.621555
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Note: Only part of the references are listed.- Functional Dynamics of Neutrophils After Ischemic Stroke
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- Extracellular ASC exacerbated the recurrent ischemic stroke in an NLRP3-dependent manner
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- NLRP3 inflammasome as a potential treatment in ischemic stroke concomitant with diabetes
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- Helicobacter pylori Exploits the NLRC4 Inflammasome to Dampen Host Defenses
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- Normal and Pathological Tau Uptake Mediated by M1/M3 Muscarinic Receptors Promotes Opposite Neuronal Changes
- (2019) Viktoriya Morozova et al. Frontiers in Cellular Neuroscience
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- (2019) J. Michelle Kahlenberg et al. Arthritis & Rheumatology
- Update of inflammasome activation in microglia/macrophage in aging and aging‐related disease
- (2019) Meng‐yan Hu et al. CNS Neuroscience & Therapeutics
- TLR-stimulated IRAKM activates caspase-8 inflammasome in microglia and promotes neuroinflammation
- (2018) Cun-Jin Zhang et al. JOURNAL OF CLINICAL INVESTIGATION
- NLR members NLRC4 and NLRP3 mediate sterile inflammasome activation in microglia and astrocytes
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- NLRP3 inflammasome: a promising target in ischemic stroke
- (2016) Li Gao et al. INFLAMMATION RESEARCH
- Pericyte MyD88 and IRAK4 control inflammatory and fibrotic responses to tissue injury
- (2016) Irina A. Leaf et al. JOURNAL OF CLINICAL INVESTIGATION
- Inflammasomes of the intestinal epithelium
- (2015) Mikael E. Sellin et al. TRENDS IN IMMUNOLOGY
- Reliability of Two Diameters Method in Determining Acute Infarct Size. Validation as New Imaging Biomarker
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- Temporal and Spatial Dynamics of Cerebral Immune Cell Accumulation in Stroke
- (2009) Mathias Gelderblom et al. STROKE
- Hsp27 Protects against Ischemic Brain Injury via Attenuation of a Novel Stress-Response Cascade Upstream of Mitochondrial Cell Death Signaling
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