Pluripotent stem cell model of early hematopoiesis in Down syndrome reveals quantitative effects of short-form GATA1 protein on lineage specification
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Title
Pluripotent stem cell model of early hematopoiesis in Down syndrome reveals quantitative effects of short-form GATA1 protein on lineage specification
Authors
Keywords
Cloning, Flow cytometry, Bone marrow cells, Cell differentiation, Cell staining, Hyperexpression techniques, Megakaryocytes, Stem cells
Journal
PLoS One
Volume 16, Issue 3, Pages e0247595
Publisher
Public Library of Science (PLoS)
Online
2021-03-30
DOI
10.1371/journal.pone.0247595
References
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- Transient Abnormal Myelopoiesis and AML in Down Syndrome: an Update
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- (2013) I. Roberts et al. BLOOD
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- The landscape of somatic mutations in Down syndrome–related myeloid disorders
- (2013) Kenichi Yoshida et al. NATURE GENETICS
- Natural history of transient myeloproliferative disorder clinically diagnosed in Down syndrome neonates: a report from the Children's Oncology Group Study A2971
- (2011) A. S. Gamis et al. BLOOD
- Down syndrome and GATA1 mutations in transient abnormal myeloproliferative disorder: mutation classes correlate with progression to myeloid leukemia
- (2010) R. Kanezaki et al. BLOOD
- Acute megakaryoblastic leukaemia (AMKL) and transient myeloproliferative disorder (TMD) in Down syndrome: a multi-step model of myeloid leukaemogenesis
- (2009) Anindita Roy et al. BRITISH JOURNAL OF HAEMATOLOGY
- Treatment and prognostic impact of transient leukemia in neonates with Down syndrome
- (2008) J.-H. Klusmann et al. BLOOD
- Risk factors for early death in neonates with Down syndrome and transient leukaemia
- (2008) Hideki Muramatsu et al. BRITISH JOURNAL OF HAEMATOLOGY
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