Cooperative Binding of ETS2 and NFAT Link Erk1/2 and Calcineurin Signaling in the Pathogenesis of Cardiac Hypertrophy

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ERK: A Key Player in the Pathophysiology of Cardiac Hypertrophy

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Calcineurin signaling in the heart: The importance of time and place

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A circular RNA protects the heart from pathological hypertrophy and heart failure by targeting miR-223

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Inhibition of Hypertrophy Is a Good Therapeutic Strategy in Ventricular Pressure Overload

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Spliced X-Box Binding Protein 1 Couples the Unfolded Protein Response to Hexosamine Biosynthetic Pathway

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MURC/Cavin-4 facilitates recruitment of ERK to caveolae and concentric cardiac hypertrophy induced by  1-adrenergic receptors

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Ets-1 facilitates nuclear entry of NFAT proteins and their recruitment to the IL-2 promoter

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Extracellular signal-regulated kinase 1/2 (ERK1/2) signaling in cardiac hypertrophy

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The CCAAT/Enhancer Binding Protein β (C/EBPβ) Cooperates with NFAT to Control Expression of the Calcineurin Regulatory ProteinRCAN1–4

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Ets1 and Ets2 are required for endothelial cell survival during embryonic angiogenesis

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Negative-feedback regulation of FGF signalling by DUSP6/MKP-3 is driven by ERK1/2 and mediated by Ets factor binding to a conserved site within theDUSP6/MKP-3gene promoter

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A new type of ERK1/2 autophosphorylation causes cardiac hypertrophy

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Cardiac Plasticity

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