The roles of STAT6 in regulating B cell fate, activation, and function
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Title
The roles of STAT6 in regulating B cell fate, activation, and function
Authors
Keywords
STAT6, B cells, IL-4
Journal
IMMUNOLOGY LETTERS
Volume -, Issue -, Pages -
Publisher
Elsevier BV
Online
2021-03-02
DOI
10.1016/j.imlet.2021.02.006
References
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Note: Only part of the references are listed.- The signal transducer and activator of transcription 6 (STAT-6) mediates Th2 inflammation and tissue damage in a murine model of peanut-induced food allergy
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- Modulation of the humoral immune response by constitutively active STAT6 expression in murine B cells
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- Nonredundant Roles of IL-21 and IL-4 in the Phased Initiation of Germinal Center B Cells and Subsequent Self-Renewal Transitions
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- IL4 and IL21 cooperate to induce the high Bcl6 protein level required for germinal center formation
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- Germinal Center B Cell Dynamics
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- STAT6-mediated BCL6 repression in primary mediastinal B-cell lymphoma (PMBL)
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- B-cell-intrinsic STAT6 signaling controls germinal center formation
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- IL-4 Regulates Bim Expression and Promotes B Cell Maturation in Synergy with BAFF Conferring Resistance to Cell Death at Negative Selection Checkpoints
- (2014) A. Granato et al. JOURNAL OF IMMUNOLOGY
- γδ T cells affect IL-4 production and B-cell tolerance
- (2014) Yafei Huang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Live-Cell Imaging of the Association of STAT6-GFP with Mitochondria
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- Loss of STAT6 promotes autoimmune disease and atopy on a susceptible genetic background
- (2012) Maverick Lau et al. JOURNAL OF AUTOIMMUNITY
- Mitochondrial Localization of Telomeric Protein TIN2 Links Telomere Regulation to Metabolic Control
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- Characterization of STAT6 Target Genes in Human B Cells and Lung Epithelial Cells
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- Transcriptional regulation by STAT6
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- GANP suppresses the arginine methyltransferase PRMT5 regulating IL-4-mediated STAT6-signaling to IgE production in B cells
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- PTP1B is a negative regulator of interleukin 4-induced STAT6 signaling
- (2008) X. Lu et al. BLOOD
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