- Home
- Publications
- Publication Search
- Publication Details
Title
Matrix Metalloproteinase-12 Is Required for Granuloma Progression
Authors
Keywords
-
Journal
Frontiers in Immunology
Volume 11, Issue -, Pages -
Publisher
Frontiers Media SA
Online
2020-09-18
DOI
10.3389/fimmu.2020.553949
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Environmental Risk Factors for Sarcoidosis
- (2020) Marc A. Judson Frontiers in Immunology
- Alveolar Macrophage ABCG1 Deficiency Promotes Pulmonary Granulomatous Inflammation
- (2019) Matthew McPeek et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- PPARγ Deficiency Exacerbates Fibrotic Response to Mycobacteria Peptide in Murine Sarcoidosis Model
- (2019) Anagha Malur et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- More Than Just Attractive: How CCL2 Influences Myeloid Cell Behavior Beyond Chemotaxis
- (2019) Martha Gschwandtner et al. Frontiers in Immunology
- Transcriptional survey of alveolar macrophages in a murine model of chronic granulomatous inflammation reveals common themes with human sarcoidosis
- (2018) Arjun Mohan et al. AMERICAN JOURNAL OF PHYSIOLOGY-LUNG CELLULAR AND MOLECULAR PHYSIOLOGY
- Sarcoidosis deaths in the United States: 1999–2016
- (2018) Gregory D. Kearney et al. RESPIRATORY MEDICINE
- Inhibition of Tissue Matrix Metalloproteinases Interferes with Mycobacterium tuberculosis-Induced Granuloma Formation and Reduces Bacterial Load in a Human Lung Tissue Model
- (2017) Venkata R. Parasa et al. Frontiers in Microbiology
- IFN-γ–Producing T-Helper 17.1 Cells Are Increased in Sarcoidosis and Are More Prevalent than T-Helper Type 1 Cells
- (2016) Joris Ramstein et al. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE
- Matrix metalloproteinases in destructive lung disease
- (2015) A. McGarry Houghton MATRIX BIOLOGY
- Diverse functions of matrix metalloproteinases during fibrosis
- (2014) M. Giannandrea et al. Disease Models & Mechanisms
- Carbon Nanotube-Induced Pulmonary Granulomatous Disease: Twist1 and Alveolar Macrophage M1 Activation
- (2013) Barbara Barna et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- The role of PPARγ in carbon nanotube-elicited granulomatous lung inflammation
- (2013) Isham Huizar et al. RESPIRATORY RESEARCH
- Sarcoidosis-related Mortality in the United States from 1988 to 2007
- (2011) Jeffrey J. Swigris et al. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE
- Novel Murine Model of Chronic Granulomatous Lung Inflammation Elicited by Carbon Nanotubes
- (2011) Isham Huizar et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- Fibrogenic and Redox-Related but not Proinflammatory Genes are Upregulated in Lewis Rat Model of Chronic Silicosis
- (2011) Raymond J. Langley et al. JOURNAL OF TOXICOLOGY AND ENVIRONMENTAL HEALTH-PART A-CURRENT ISSUES
- Detection of endogenous cytokines in sera or in lymph nodes obtained from patients with sarcoidosis
- (2010) M. ASANO et al. CLINICAL AND EXPERIMENTAL IMMUNOLOGY
- Gene Expression Profiling Identifies MMP-12 and ADAMDEC1 as Potential Pathogenic Mediators of Pulmonary Sarcoidosis
- (2009) Elliott D. Crouser et al. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE
- Case Report: Lung Disease in World Trade Center Responders Exposed to Dust and Smoke: Carbon Nanotubes Found in the Lungs of World Trade Center Patients and Dust Samples
- (2009) Maoxin Wu et al. ENVIRONMENTAL HEALTH PERSPECTIVES
- Deletion of PPAR in Alveolar Macrophages Is Associated with a Th-1 Pulmonary Inflammatory Response
- (2009) A. Malur et al. JOURNAL OF IMMUNOLOGY
- Standardized quantification of pulmonary fibrosis in histological samples
- (2008) Ralf-Harto Hübner et al. BIOTECHNIQUES
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExplorePublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More