Change the Laminin, Change the Cardiomyocyte: Improve Untreatable Heart Failure
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Title
Change the Laminin, Change the Cardiomyocyte: Improve Untreatable Heart Failure
Authors
Keywords
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Journal
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
Volume 21, Issue 17, Pages 6013
Publisher
MDPI AG
Online
2020-08-21
DOI
10.3390/ijms21176013
References
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Related references
Note: Only part of the references are listed.- Type IV collagen conforms to the organization of polylaminin adsorbed on planar substrata
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- Epidemiology of heart failure with preserved ejection fraction
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- 2016 ESC Guidelines for the diagnosis and treatment of acute and chronic heart failure
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- The role of titin and extracellular matrix remodelling in heart failure with preserved ejection fraction
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- Deleting titin’s I-band/A-band junction reveals critical roles for titin in biomechanical sensing and cardiac function
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- Effect of Phosphodiesterase-5 Inhibition on Exercise Capacity and Clinical Status in Heart Failure With Preserved Ejection Fraction
- (2013) Margaret M. Redfield et al. JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION
- RBM20, a gene for hereditary cardiomyopathy, regulates titin splicing
- (2012) Wei Guo et al. NATURE MEDICINE
- Polylaminin, a polymeric form of laminin, promotes regeneration after spinal cord injury
- (2010) Karla Menezes et al. FASEB JOURNAL
- The Giant Protein Titin as an Integrator of Myocyte Signaling Pathways
- (2010) Wolfgang A. Linke et al. PHYSIOLOGY
- Hypophosphorylation of the Stiff N2B Titin Isoform Raises Cardiomyocyte Resting Tension in Failing Human Myocardium
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