Bacteria-derived long chain fatty acid exhibits anti-inflammatory properties in colitis

Endogenous murine microbiota member Faecalibaculum rodentium and its human homologue protect from intestinal tumour growth

(2020) Elena Zagato et al.   Nature Microbiology

The gut microbiome influences host endocrine functions

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The administration of Escherichia coli Nissle 1917 ameliorates irinotecan–induced intestinal barrier dysfunction and gut microbial dysbiosis in mice

(2019) Yurong Wang et al.   LIFE SCIENCES

Deciphering the interplay between the genotoxic and probiotic activities of Escherichia coli Nissle 1917

(2019) Clémence Massip et al.   PLoS Pathogens

Systematic review: safety of mesalazine in ulcerative colitis

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Escherichia coli Nissle 1917 restores epithelial permeability alterations induced by irritable bowel syndrome mediators

(2018) M. R. Barbaro et al.   NEUROGASTROENTEROLOGY AND MOTILITY

Expert consensus document: The International Scientific Association for Probiotics and Prebiotics (ISAPP) consensus statement on the definition and scope of prebiotics

(2017) Glenn R. Gibson et al.   Nature Reviews Gastroenterology & Hepatology

Microbiota-activated PPAR-γ signaling inhibits dysbiotic Enterobacteriaceae expansion

(2017) Mariana X. Byndloss et al.   SCIENCE

Identification of an analgesic lipopeptide produced by the probiotic Escherichia coli strain Nissle 1917

(2017) Teresa Pérez-Berezo et al.   Nature Communications

Escherichia coli strain Nissle 1917 ameliorates experimental colitis by modulating intestinal permeability, the inflammatory response and clinical signs in a faecal transplantation model

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Intestinal PPARγ signalling is required for sympathetic nervous system activation in response to caloric restriction

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Oral Tolerance Failure upon Neonatal Gut Colonization with Escherichia coli Producing the Genotoxin Colibactin

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A Novel Peroxisome Proliferator-activated Receptor (PPAR)γ Agonist 2-Hydroxyethyl 5-chloro-4,5-didehydrojasmonate Exerts Anti-Inflammatory Effects in Colitis

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Faecalicoccus acidiformans gen. nov., sp. nov., isolated from the chicken caecum, and reclassification of Streptococcus pleomorphus (Barnes et al. 1977), Eubacterium biforme (Eggerth 1935) and Eubacterium cylindroides (Cato et al. 1974) as Faecalicoccus pleomorphus comb. nov., Holdemanella biformis gen. nov., comb. nov. and Faecalitalea cylindroides gen. nov., comb. nov., respectively, within the family Erysipelotrichaceae

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Microbiome of prebiotic-treated mice reveals novel targets involved in host response during obesity

(2014) Amandine Everard et al.   ISME Journal

Pro-resolving lipid mediators are leads for resolution physiology

(2014) Charles N. Serhan   NATURE

A role for the gut microbiota in IBS

(2014) Stephen M. Collins   Nature Reviews Gastroenterology & Hepatology

Dysbiotic Events in Gut Microbiota: Impact on Human Health

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PPARs are a unique set of fatty acid regulated transcription factors controlling both lipid metabolism and inflammation

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Responses of Gut Microbiota and Glucose and Lipid Metabolism to Prebiotics in Genetic Obese and Diet-Induced Leptin-Resistant Mice

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Recommendations for Probiotic Use—2011 Update

(2011) Martin H. Floch et al.   JOURNAL OF CLINICAL GASTROENTEROLOGY

Comparative Gene Expression Profiles Induced by PPARγ and PPARα/γ Agonists in Human Hepatocytes

(2011) Alexandra Rogue et al.   PLoS One

Optimizing clinical use of mesalazine (5-aminosalicylic acid) in inflammatory bowel disease

(2011) Chadwick Williams et al.   Therapeutic Advances in Gastroenterology

The K5 Capsule of Escherichia coli Strain Nissle 1917 Is Important in Stimulating Expression of Toll-Like Receptor 5, CD14, MyD88, and TRIF Together with the Induction of Interleukin-8 Expression via the Mitogen-Activated Protein Kinase Pathway in Epithelial Cells

(2010) M. Hafez et al.   INFECTION AND IMMUNITY

Pharmacological correction of a defect in PPAR-γ signaling ameliorates disease severity in Cftr-deficient mice

(2010) Gregory S Harmon et al.   NATURE MEDICINE

Changes in gut microbiota control inflammation in obese mice through a mechanism involving GLP-2-driven improvement of gut permeability

(2009) P. D. Cani et al.   GUT