Neutrophil autophagy during human active tuberculosis is modulated by SLAMF1
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Title
Neutrophil autophagy during human active tuberculosis is modulated by SLAMF1
Authors
Keywords
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Journal
Autophagy
Volume -, Issue -, Pages 1-10
Publisher
Informa UK Limited
Online
2020-09-20
DOI
10.1080/15548627.2020.1825273
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- (2018) Yasuaki Aratani ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS
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- (2017) Irina V. Lyadova MEDIATORS OF INFLAMMATION
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- Reactive Oxygen Species and Neutrophil Function
- (2016) Christine C. Winterbourn et al. Annual Review of Biochemistry
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- (2014) Ana I Rovetta et al. Autophagy
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- (2010) A. M. Cooper JOURNAL OF LEUKOCYTE BIOLOGY
- An interferon-inducible neutrophil-driven blood transcriptional signature in human tuberculosis
- (2010) Matthew P. R. Berry et al. NATURE
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- (2010) Scott B Berger et al. NATURE IMMUNOLOGY
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