Effects and Mechanism of lncRNA CRNDE on Sepsis-Induced Acute Kidney Injury
Published 2020 View Full Article
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Title
Effects and Mechanism of lncRNA CRNDE on Sepsis-Induced Acute Kidney Injury
Authors
Keywords
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Journal
Analytical Cellular Pathology
Volume 2020, Issue -, Pages 1-8
Publisher
Hindawi Limited
Online
2020-05-02
DOI
10.1155/2020/8576234
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- (2019) Hongxi Li et al. LIFE SCIENCES
- Down-regulation of LncRNA CRNDE aggravates kidney injury via increasing MiR-181a-5p in sepsis
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- Knockdown CRNDE alleviates LPS-induced inflammation injury via FOXM1 in WI-38 cells
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- LncRNA MALAT1 regulates sepsis-induced cardiac inflammation and dysfunction via interaction with miR-125b and p38 MAPK/NFκB
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- MicroRNA-146a protects against LPS-induced organ damage by inhibiting Notch1 in macrophage
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- Noncoding RNAs in acute kidney injury
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- Overexpression of miR‑146a blocks the effect of LPS on RANKL‑induced osteoclast differentiation
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- Mechanism of long non-coding RNA MALAT1 in lipopolysaccharide-induced acute kidney injury is mediated by the miR-146a/NF-κB signaling pathway
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- CRNDE, a long-noncoding RNA, promotes glioma cell growth and invasion through mTOR signaling
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