Gut Microbiota Contributes to Spontaneous Colitis in E3 Ligase Itch-Deficient Mice
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Title
Gut Microbiota Contributes to Spontaneous Colitis in E3 Ligase Itch-Deficient Mice
Authors
Keywords
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Journal
JOURNAL OF IMMUNOLOGY
Volume 204, Issue 8, Pages 2277-2284
Publisher
The American Association of Immunologists
Online
2020-03-14
DOI
10.4049/jimmunol.1701478
References
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Related references
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- Itch inhibits IL-17-mediated colon inflammation and tumorigenesis by ROR-γt ubiquitination
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- The NOD-Like Receptor NLRP12 Attenuates Colon Inflammation and Tumorigenesis
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- NLRP6 Inflammasome Regulates Colonic Microbial Ecology and Risk for Colitis
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- (2010) Wendy S. Garrett et al. CELL
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- (2010) Janelle C. Arthur et al. INFLAMMATORY BOWEL DISEASES
- Global patterns of 16S rRNA diversity at a depth of millions of sequences per sample
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- (2009) Lianne K.P.M. Brakenhoff et al. Journal of Crohns & Colitis
- A human colonic commensal promotes colon tumorigenesis via activation of T helper type 17 T cell responses
- (2009) Shaoguang Wu et al. NATURE MEDICINE
- Inflammatory Bowel Disease
- (2009) Clara Abraham et al. NEW ENGLAND JOURNAL OF MEDICINE
- Evolution of Mammals and Their Gut Microbes
- (2008) R. E. Ley et al. SCIENCE
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