A blast without power – cell death induced by the tuberculosis-necrotizing toxin fails to elicit adequate immune responses

Necroptotic cells release find-me signal and are engulfed without proinflammatory cytokine production

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The tuberculosis necrotizing toxin kills macrophages by hydrolyzing NAD

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The tuberculosis necrotizing toxin kills macrophages by hydrolyzing NAD

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Antiproliferative Activity of IL-27 on Melanoma

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An outer membrane channel protein of Mycobacterium tuberculosis with exotoxin activity

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The Progression of Cell Death Affects the Rejection of Allogeneic Tumors in Immune-Competent Mice – Implications for Cancer Therapy

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Sustained tumour eradication after induced caspase-3 activation and synchronous tumour apoptosis requires an intact host immune response

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Molecular mechanisms of ATP secretion during immunogenic cell death

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Stress hormones promote growth of B16-F10 melanoma metastases: an interleukin 6- and glutathione-dependent mechanism

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IL-27 in tumor immunity and immunotherapy

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Clearing the Dead: Apoptotic Cell Sensing, Recognition, Engulfment, and Digestion

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Colourful death: Six-parameter classification of cell death by flow cytometry—Dead cells tell tales

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Mycobacterium tuberculosis Inhibits Neutrophil Apoptosis, Leading to Delayed Activation of Naive CD4 T cells

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Mycobacterium tuberculosis Induces an Atypical Cell Death Mode to Escape from Infected Macrophages

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IL-27 increases the proliferation and effector functions of human naïve CD8+ T lymphocytes and promotes their development into Tc1 cells

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Cell death in the neighbourhood: direct microenvironmental effects of apoptosis in normal and neoplastic tissues

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The role of defective clearance of apoptotic cells in systemic autoimmunity

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Nucleotides released by apoptotic cells act as a find-me signal to promote phagocytic clearance

(2009) Michael R. Elliott et al.   NATURE

Living on the edge: inhibition of host cell apoptosis byMycobacterium tuberculosis

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Executioner caspase-3 and caspase-7 are functionally distinct proteases

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IL-27-Mediated Activation of Natural Killer Cells and Inflammation Produced Antitumour Effects for Human Oesophageal Carcinoma Cells

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