Increased expression of SIRT2 is a novel marker of cellular senescence and is dependent on wild type p53 status
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Title
Increased expression of SIRT2 is a novel marker of cellular senescence and is dependent on wild type p53 status
Authors
Keywords
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Journal
CELL CYCLE
Volume 15, Issue 14, Pages 1883-1897
Publisher
Informa UK Limited
Online
2016-05-27
DOI
10.1080/15384101.2016.1189041
References
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- Differential expression of sirtuins in the aging rat brain
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- Molecular characterization of collaborator of ARF (CARF) as a DNA damage response and cell cycle checkpoint regulatory protein
- (2014) Rumani Singh et al. EXPERIMENTAL CELL RESEARCH
- α-Fucosidase as a novel convenient biomarker for cellular senescence
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- Intracellular distribution of human SIRT7 and mapping of the nuclear/nucleolar localization signal
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- Lamin B1 fluctuations have differential effects on cellular proliferation and senescence
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- Lamin B1 loss is a senescence-associated biomarker
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- Deacetylation of H4-K16Ac and heterochromatin assembly in senescence
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- SIRT2 as a Therapeutic Target for Age-Related Disorders
- (2012) Rita Machado de Oliveira et al. Frontiers in Pharmacology
- Senescence-associated heterochromatin foci are dispensable for cellular senescence, occur in a cell type- and insult-dependent manner and follow expression of p16ink4a
- (2011) Martin Kosar et al. CELL CYCLE
- The Sirtuin 2 microtubule deacetylase is an abundant neuronal protein that accumulates in the aging CNS
- (2011) Michele M. Maxwell et al. HUMAN MOLECULAR GENETICS
- Four faces of cellular senescence
- (2011) Francis Rodier et al. JOURNAL OF CELL BIOLOGY
- PGC-1α, a Key Modulator of p53, Promotes Cell Survival upon Metabolic Stress
- (2011) Nirmalya Sen et al. MOLECULAR CELL
- Absence of effects of Sir2 overexpression on lifespan in C. elegans and Drosophila
- (2011) Camilla Burnett et al. NATURE
- p53 and p16INK4A independent induction of senescence by chromatin-dependent alteration of S-phase progression
- (2011) Alexandre Prieur et al. Nature Communications
- A novel type of cellular senescence that can be enhanced in mouse models and human tumor xenografts to suppress prostate tumorigenesis
- (2010) Andrea Alimonti et al. JOURNAL OF CLINICAL INVESTIGATION
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