Lamin B1 fluctuations have differential effects on cellular proliferation and senescence
Published 2013 View Full Article
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Title
Lamin B1 fluctuations have differential effects on cellular proliferation and senescence
Authors
Keywords
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Journal
JOURNAL OF CELL BIOLOGY
Volume 200, Issue 5, Pages 605-617
Publisher
Rockefeller University Press
Online
2013-02-26
DOI
10.1083/jcb.201206121
References
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Related references
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- Regulation of prelamin A but not lamin C by miR-9, a brain-specific microRNA
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- The role of nuclear lamin B1 in cell proliferation and senescence
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- Deficiencies in lamin B1 and lamin B2 cause neurodevelopmental defects and distinct nuclear shape abnormalities in neurons
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- Mouse B-Type Lamins Are Required for Proper Organogenesis But Not by Embryonic Stem Cells
- (2011) Y. Kim et al. SCIENCE
- A Human iPSC Model of Hutchinson Gilford Progeria Reveals Vascular Smooth Muscle and Mesenchymal Stem Cell Defects
- (2010) Jinqiu Zhang et al. Cell Stem Cell
- Role of progerin-induced telomere dysfunction in HGPS premature cellular senescence
- (2010) E. K. Benson et al. JOURNAL OF CELL SCIENCE
- Abnormal development of the cerebral cortex and cerebellum in the setting of lamin B2 deficiency
- (2010) C. Coffinier et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Diseases of the Nuclear Envelope
- (2010) H. J. Worman et al. Cold Spring Harbor Perspectives in Biology
- miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination
- (2009) S.-T. Lin et al. Disease Models & Mechanisms
- A family with autosomal dominant leukodystrophy linked to 5q23.2-q23.3 without lamin B1 mutations
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- Lamin B1 controls oxidative stress responses via Oct-1
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- Telomere length in Hutchinson-Gilford Progeria Syndrome
- (2009) Michelle L. Decker et al. MECHANISMS OF AGEING AND DEVELOPMENT
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- (2009) P. Taimen et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- The integrity of a lamin-B1-dependent nucleoskeleton is a fundamental determinant of RNA synthesis in human cells
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- Suppression of Proliferative Defects Associated with Processing-defective Lamin A Mutants by hTERT or Inactivation of p53
- (2008) Brian A. Kudlow et al. MOLECULAR BIOLOGY OF THE CELL
- Domain organization of human chromosomes revealed by mapping of nuclear lamina interactions
- (2008) Lars Guelen et al. NATURE
- Loss of nucleoplasmic LAP2α–lamin A complexes causes erythroid and epidermal progenitor hyperproliferation
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