Associations between brain inflammatory profiles and human neuropathology are altered based on apolipoprotein E ε4 genotype
Published 2020 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Associations between brain inflammatory profiles and human neuropathology are altered based on apolipoprotein E ε4 genotype
Authors
Keywords
-
Journal
Scientific Reports
Volume 10, Issue 1, Pages -
Publisher
Springer Science and Business Media LLC
Online
2020-02-19
DOI
10.1038/s41598-020-59869-5
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Pro - and anti-inflammatory cytokines in the CSF of Alzheimer´s disease patients and their correlation with cognitive decline
- (2019) R. Taipa et al. NEUROBIOLOGY OF AGING
- Plasma tau correlates with basal forebrain atrophy rates in people at risk for Alzheimer disease
- (2019) Enrica Cavedo et al. NEUROLOGY
- Role of inflammatory molecules in the Alzheimer's disease progression and diagnosis
- (2017) Eva Bagyinszky et al. JOURNAL OF THE NEUROLOGICAL SCIENCES
- ApoE4 markedly exacerbates tau-mediated neurodegeneration in a mouse model of tauopathy
- (2017) Yang Shi et al. NATURE
- Anti-inflammatory (M2) macrophage media reduce transmission of oligomeric amyloid beta in differentiated SH-SY5Y cells
- (2017) Valerie Sackmann et al. NEUROBIOLOGY OF AGING
- Alzheimer disease brain atrophy subtypes are associated with cognition and rate of decline
- (2017) Shannon L. Risacher et al. NEUROLOGY
- Microglial immunophenotype in dementia with Alzheimer’s pathology
- (2016) Thais Minett et al. Journal of Neuroinflammation
- Inflammatory Response in the CNS: Friend or Foe?
- (2016) Marta Sochocka et al. MOLECULAR NEUROBIOLOGY
- Genes associated with Alzheimer's disease: an overview and current status
- (2016) Mohan Giri et al. Clinical Interventions in Aging
- The dual roles of cytokines in Alzheimer’s disease: update on interleukins, TNF-α, TGF-β and IFN-γ
- (2016) Cong Zheng et al. Translational Neurodegeneration
- Microglial neuroinflammation contributes to tau accumulation in chronic traumatic encephalopathy
- (2016) Jonathan D. Cherry et al. Acta Neuropathologica Communications
- Beta-amyloid deposition in chronic traumatic encephalopathy
- (2015) Thor D. Stein et al. ACTA NEUROPATHOLOGICA
- Reactive microglia drive tau pathology and contribute to the spreading of pathological tau in the brain
- (2015) Nicole Maphis et al. BRAIN
- Cohort Profile: The Framingham Heart Study (FHS): overview of milestones in cardiovascular epidemiology
- (2015) Connie W Tsao et al. INTERNATIONAL JOURNAL OF EPIDEMIOLOGY
- APOE-modulated Aβ-induced neuroinflammation in Alzheimer's disease: current landscape, novel data, and future perspective
- (2015) Leon M. Tai et al. JOURNAL OF NEUROCHEMISTRY
- Activated human microglia stimulate neuroblastoma cells to upregulate production of beta amyloid protein and tau: implications for Alzheimer's disease pathogenesis
- (2015) Moonhee Lee et al. NEUROBIOLOGY OF AGING
- Il10 Deficiency Rebalances Innate Immunity to Mitigate Alzheimer-Like Pathology
- (2015) Marie-Victoire Guillot-Sestier et al. NEURON
- IL-10 Alters Immunoproteostasis in APP Mice, Increasing Plaque Burden and Worsening Cognitive Behavior
- (2015) Paramita Chakrabarty et al. NEURON
- Assessing clinicopathological correlation in chronic traumatic encephalopathy: rationale and methods for the UNITE study
- (2015) Jesse Mez et al. Alzheimers Research & Therapy
- Association Between Neuropathology and Brain Volume in The Framingham Heart Study
- (2014) Berneet Kaur et al. ALZHEIMER DISEASE & ASSOCIATED DISORDERS
- A critical analysis of the ‘amyloid cascade hypothesis’
- (2014) Richard A. Armstrong FOLIA NEUROPATHOLOGICA
- Human APOE4 increases microglia reactivity at Aβ plaques in a mouse model of Aβ deposition
- (2014) Gustavo A Rodriguez et al. Journal of Neuroinflammation
- Tau Overexpression Impacts a Neuroinflammation Gene Expression Network Perturbed in Alzheimer’s Disease
- (2014) Paul D. Wes et al. PLoS One
- Expression of Tau40 Induces Activation of Cultured Rat Microglial Cells
- (2013) Lu Wang et al. PLoS One
- A Review: Inflammatory Process in Alzheimer's Disease, Role of Cytokines
- (2012) Jose Miguel Rubio-Perez et al. TheScientificWorldJOURNAL
- National Institute on Aging–Alzheimer’s Association guidelines for the neuropathologic assessment of Alzheimer’s disease: a practical approach
- (2011) Thomas J. Montine et al. ACTA NEUROPATHOLOGICA
- AAV serotype 2/1-mediated gene delivery of anti-inflammatory interleukin-10 enhances neurogenesis and cognitive function in APP+PS1 mice
- (2011) T Kiyota et al. GENE THERAPY
- Stages of the Pathologic Process in Alzheimer Disease: Age Categories From 1 to 100 Years
- (2011) Heiko Braak et al. JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY
- Apolipoprotein E in Alzheimer's disease and other neurological disorders
- (2011) Philip B Verghese et al. LANCET NEUROLOGY
- Hippocampal expression of murine TNFα results in attenuation of amyloid deposition in vivo
- (2011) Paramita Chakrabarty et al. Molecular Neurodegeneration
- CNS expression of anti-inflammatory cytokine interleukin-4 attenuates Alzheimer’s disease-like pathogenesis in APP+PS1 bigenic mice
- (2010) Tomomi Kiyota et al. FASEB JOURNAL
- The Genetics of Alzheimer Disease: Back to the Future
- (2010) Lars Bertram et al. NEURON
- Interferon-γ Differentially Affects Alzheimer’s Disease Pathologies and Induces Neurogenesis in Triple Transgenic-AD Mice
- (2009) Michael A. Mastrangelo et al. AMERICAN JOURNAL OF PATHOLOGY
- Microglial activation in brain lesions with tau deposits: Comparison of human tauopathies and tau transgenic mice TgTauP301L
- (2008) Atsushi Sasaki et al. BRAIN RESEARCH
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started