Resveratrol Suppresses Prostate Cancer Epithelial Cell Scatter/Invasion by Targeting Inhibition of Hepatocyte Growth Factor (HGF) Secretion by Prostate Stromal Cells and Upregulation of E-cadherin by Prostate Cancer Epithelial Cells
Published 2020 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Resveratrol Suppresses Prostate Cancer Epithelial Cell Scatter/Invasion by Targeting Inhibition of Hepatocyte Growth Factor (HGF) Secretion by Prostate Stromal Cells and Upregulation of E-cadherin by Prostate Cancer Epithelial Cells
Authors
Keywords
-
Journal
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
Volume 21, Issue 5, Pages 1760
Publisher
MDPI AG
Online
2020-03-04
DOI
10.3390/ijms21051760
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Epithelial-Mesenchymal Plasticity in Cancer Progression and Metastasis
- (2019) Wei Lu et al. DEVELOPMENTAL CELL
- Restoring E-cadherin expression by natural compounds for anticancer therapies in genital and urinary cancers
- (2019) Yizuo Song et al. Molecular Therapy-Oncolytics
- Thiosemicarbazones suppress expression of the c-Met oncogene by mechanisms involving lysosomal degradation and intracellular shedding.
- (2019) Kyung Chan Park et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Aberrant activation of hepatocyte growth factor/MET signaling promotes β-catenin–mediated prostatic tumorigenesis
- (2019) Joseph Aldahl et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Factors involved in cancer metastasis: a better understanding to “seed and soil” hypothesis
- (2017) Qiang Liu et al. Molecular Cancer
- Revisiting Seed and Soil: Examining the Primary Tumor and Cancer Cell Foraging in Metastasis
- (2017) Amber E. de Groot et al. MOLECULAR CANCER RESEARCH
- Stromal and epithelial transcriptional map of initiation progression and metastatic potential of human prostate cancer
- (2017) Svitlana Tyekucheva et al. Nature Communications
- Curcumin mediates reversion of HGF-induced epithelial-mesenchymal transition via inhibition of c-Met expression in DU145 cells
- (2015) HUI-JUN HU et al. Oncology Letters
- Hepatocyte growth factor increases the invasive potential of PC-3 human prostate cancer cells via an ERK/MAPK and Zeb-1 signaling pathway
- (2015) YILI HAN et al. Oncology Letters
- TLE1 promotes EMT in A549 lung cancer cells through suppression of E-cadherin
- (2014) Xin Yao et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Redefining micrometastasis in prostate cancer - a comparison of circulating prostate cells, bone marrow disseminated tumor cells and micrometastasis: Implications in determining local or systemic treatment for biochemical failure after radical prostatectomy
- (2012) N.P. MURRAY et al. INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE
- Steps in prostate cancer progression that lead to bone metastasis
- (2011) Jung-Kang Jin et al. INTERNATIONAL JOURNAL OF CANCER
- The seed and soil hypothesis revisited-The role of tumor-stroma interactions in metastasis to different organs
- (2011) Robert R. Langley et al. INTERNATIONAL JOURNAL OF CANCER
- Diosgenin Suppresses Hepatocyte Growth Factor (HGF)-Induced Epithelial–Mesenchymal Transition by Down-regulation of Mdm2 and Vimentin
- (2011) Hsiang-Yu Chang et al. JOURNAL OF AGRICULTURAL AND FOOD CHEMISTRY
- The polyphenol epigallocatechin-3-gallate affects lipid rafts to block activation of the c-Met receptor in prostate cancer cells
- (2010) Damian Duhon et al. MOLECULAR CARCINOGENESIS
- Uptake of resveratrol and role of resveratrol-targeting protein, quinone reductase 2, in normally cultured human prostate cells
- (2009) Tze-Chen Hsieh ASIAN JOURNAL OF ANDROLOGY
- Tumor–stroma co-evolution in prostate cancer progression and metastasis
- (2009) Sajni Josson et al. SEMINARS IN CELL & DEVELOPMENTAL BIOLOGY
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExplorePublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More