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Title
DNA Damage and DNA Damage Response in Chronic Myeloid Leukemia
Authors
Keywords
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Journal
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
Volume 21, Issue 4, Pages 1177
Publisher
MDPI AG
Online
2020-02-11
DOI
10.3390/ijms21041177
References
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Note: Only part of the references are listed.- Diagnostic performance of the molecular BCR-ABL1 monitoring system may impact on inclusion of CML patients in stopping trials
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- (2017) Henning D. Popp et al. LEUKEMIA RESEARCH
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- (2017) Henning D. Popp et al. Jove-Journal of Visualized Experiments
- 53BP1 promotes microhomology-mediated end-joining in G1-phase cells
- (2015) Xiahui Xiong et al. NUCLEIC ACIDS RESEARCH
- European LeukemiaNet recommendations for the management of chronic myeloid leukemia: 2013
- (2013) M. Baccarani et al. BLOOD
- 53BP1 Mediates Productive and Mutagenic DNA Repair through Distinct Phosphoprotein Interactions
- (2013) Elsa Callen et al. CELL
- RIF1 Is Essential for 53BP1-Dependent Nonhomologous End Joining and Suppression of DNA Double-Strand Break Resection
- (2013) J. Ross Chapman et al. MOLECULAR CELL
- Double strand break repair functions of histone H2AX
- (2013) Ralph Scully et al. MUTATION RESEARCH-FUNDAMENTAL AND MOLECULAR MECHANISMS OF MUTAGENESIS
- DNA damage-induced cell death: From specific DNA lesions to the DNA damage response and apoptosis
- (2012) Wynand P. Roos et al. CANCER LETTERS
- Targeting abnormal DNA double-strand break repair in tyrosine kinase inhibitor-resistant chronic myeloid leukemias
- (2012) L A Tobin et al. ONCOGENE
- Genomic Instability in Chronic Myeloid Leukemia: Targets for Therapy?
- (2012) N. Muvarak et al. Current Hematologic Malignancy Reports
- Impact of additional cytogenetic aberrations at diagnosis on prognosis of CML: long-term observation of 1151 patients from the randomized CML Study IV
- (2011) A. Fabarius et al. BLOOD
- BCR/ABL Inhibits Mismatch Repair to Protect from Apoptosis and Induce Point Mutations
- (2008) T. Stoklosa et al. CANCER RESEARCH
- BCR/ABL and Other Kinases from Chronic Myeloproliferative Disorders Stimulate Single-Strand Annealing, an Unfaithful DNA Double-Strand Break Repair
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- BCR/ABL promotes accumulation of chromosomal aberrations induced by oxidative and genotoxic stress
- (2008) M Koptyra et al. LEUKEMIA
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- (2008) Simone Difilippantonio et al. NATURE
- An Oncogene-Induced DNA Damage Model for Cancer Development
- (2008) T. D. Halazonetis et al. SCIENCE
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