Ca2+-dependent recruitment of voltage-gated sodium channels underlies bilirubin-induced overexcitation and neurotoxicity
Published 2019 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Ca2+-dependent recruitment of voltage-gated sodium channels underlies bilirubin-induced overexcitation and neurotoxicity
Authors
Keywords
-
Journal
Cell Death & Disease
Volume 10, Issue 10, Pages -
Publisher
Springer Science and Business Media LLC
Online
2019-10-11
DOI
10.1038/s41419-019-1979-1
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- P2Y12 regulates microglia activation and excitatory synaptic transmission in spinal lamina II neurons during neuropathic pain in rodents
- (2019) Tingting Yu et al. Cell Death & Disease
- Modulation of Cav2.3 channels by unconjugated bilirubin (UCB) – Candidate mechanism for UCB-induced neuromodulation and neurotoxicity
- (2019) Walid Albanna et al. MOLECULAR AND CELLULAR NEUROSCIENCE
- Accelerated Development of the First-Order Central Auditory Neurons With Spontaneous Activity
- (2018) Xin-Lu Yin et al. Frontiers in Molecular Neuroscience
- Bilirubin augments Ca2+ load of developing bushy neurons by targeting specific subtype of voltage-gated calcium channels
- (2017) Min Liang et al. Scientific Reports
- The role of gamma-aminobutyric acid/glycinergic synaptic transmission in mediating bilirubin-induced hyperexcitation in developing auditory neurons
- (2016) Xin-Lu Yin et al. TOXICOLOGY LETTERS
- Voltage-Gated Sodium Channels: Structure, Function, Pharmacology, and Clinical Indications
- (2015) Manuel de Lera Ruiz et al. JOURNAL OF MEDICINAL CHEMISTRY
- The clinical syndrome of bilirubin-induced neurologic dysfunction
- (2015) Vinod K. Bhutani et al. Seminars in Fetal & Neonatal Medicine
- Movement disorders due to bilirubin toxicity
- (2015) Jessica Rose et al. Seminars in Fetal & Neonatal Medicine
- Riluzole is a Promising Pharmacological Inhibitor of Bilirubin-Induced Excitotoxicity in the Ventral Cochlear Nucleus
- (2014) Guo-Ying Han et al. CNS Neuroscience & Therapeutics
- Age-Related Homeostatic Midchannel Proteolysis of Neuronal L-type Voltage-Gated Ca2+ Channels
- (2014) Ioannis E. Michailidis et al. NEURON
- Bilirubin-Induced Neurologic Damage — Mechanisms and Management Approaches
- (2013) Jon F. Watchko et al. NEW ENGLAND JOURNAL OF MEDICINE
- ERK1/2 Regulate Exocytosis through Direct Phosphorylation of the Exocyst Component Exo70
- (2012) Jinqi Ren et al. DEVELOPMENTAL CELL
- Vestibular evoked myogenic potentials in term newborn infants with severe hyperbilirubinemia
- (2012) Servet Ozkiraz et al. PEDIATRICS INTERNATIONAL
- Bilirubin facilitates depolarizing GABA/glycinergic synaptic transmission in the ventral cochlear nucleus of rats
- (2011) Chun-Yan Li et al. EUROPEAN JOURNAL OF PHARMACOLOGY
- Selective vulnerability of rat brain regions to unconjugated bilirubin
- (2011) Ana Rita Vaz et al. MOLECULAR AND CELLULAR NEUROSCIENCE
- Bilirubin enhances neuronal excitability by increasing glutamatergic transmission in the rat lateral superior olive
- (2011) Chun-Yan Li et al. TOXICOLOGY
- Calculated free bilirubin levels and neurotoxicity
- (2009) M J Daood et al. Journal of Perinatology
- Nav1.1 is predominantly expressed in nodes of Ranvier and axon initial segments
- (2008) Amandine Duflocq et al. MOLECULAR AND CELLULAR NEUROSCIENCE
Create your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create NowBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started