DKK1 promotes hepatocellular carcinoma inflammation, migration and invasion: Implication of TGF-β1
Published 2019 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
DKK1 promotes hepatocellular carcinoma inflammation, migration and invasion: Implication of TGF-β1
Authors
Keywords
-
Journal
PLoS One
Volume 14, Issue 9, Pages e0223252
Publisher
Public Library of Science (PLoS)
Online
2019-10-01
DOI
10.1371/journal.pone.0223252
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- TGF-β1 Promotes Hepatocellular Carcinoma Invasion and Metastasis via ERK Pathway-Mediated FGFR4 Expression
- (2018) Jiawei Huang et al. CELLULAR PHYSIOLOGY AND BIOCHEMISTRY
- Alpha-fetoprotein inhibits autophagy to promote malignant behaviour in hepatocellular carcinoma cells by activating PI3K/AKT/mTOR signalling
- (2018) Shanshan Wang et al. Cell Death & Disease
- Differential effects on lung and bone metastasis of breast cancer by Wnt signalling inhibitor DKK1
- (2017) Xueqian Zhuang et al. NATURE CELL BIOLOGY
- DKK1 promotes migration and invasion of non–small cell lung cancer via β-catenin signaling pathway
- (2017) Jing Zhang et al. TUMOR BIOLOGY
- Inhibition of wnt/β-catenin Signaling in Hepatocellular Carcinoma by an Antipsychotic Drug Pimozide
- (2016) Valerie Fako et al. International Journal of Biological Sciences
- Action and function of Wnt/β-catenin signaling in the progression from chronic hepatitis C to hepatocellular carcinoma
- (2016) Wenhui Wang et al. JOURNAL OF GASTROENTEROLOGY
- Genetic Landscape and Biomarkers of Hepatocellular Carcinoma
- (2015) Jessica Zucman-Rossi et al. GASTROENTEROLOGY
- Exome sequencing of hepatocellular carcinomas identifies new mutational signatures and potential therapeutic targets
- (2015) Kornelius Schulze et al. NATURE GENETICS
- TGFβSignaling in Tumor Initiation, Epithelial-to-Mesenchymal Transition, and Metastasis
- (2015) Panagiotis Papageorgis Journal of Oncology
- Transforming Growth Factor-Beta and Matrix Metalloproteinases: Functional Interactions in Tumor Stroma-Infiltrating Myeloid Cells
- (2014) Jelena Krstic et al. TheScientificWorldJOURNAL
- Wnt signaling and hepatocarcinogenesis: Molecular targets for the development of innovative anticancer drugs
- (2013) Floriane Pez et al. JOURNAL OF HEPATOLOGY
- Dickkopf-1 inhibits epithelial-mesenchymal transition of colon cancer cells and contributes to colon cancer suppression
- (2012) Lisha Qi et al. CANCER SCIENCE
- Dickkopf-1 (DKK1) promotes invasion and metastasis of hepatocellular carcinoma
- (2012) Yi-Ming Tao et al. DIGESTIVE AND LIVER DISEASE
- Oncogenic β-catenin triggers an inflammatory response that determines the aggressiveness of hepatocellular carcinoma in mice
- (2012) Marie Anson et al. JOURNAL OF CLINICAL INVESTIGATION
- Dickkopf-1 and hepatocellular carcinoma
- (2012) Jung-Fa Tsai et al. LANCET ONCOLOGY
- Serum DKK1 as a protein biomarker for the diagnosis of hepatocellular carcinoma: a large-scale, multicentre study
- (2012) Qiujin Shen et al. LANCET ONCOLOGY
- Dickkopf1: A tumor suppressor or metastasis promoter?
- (2011) Mitchell E. Menezes et al. INTERNATIONAL JOURNAL OF CANCER
- Wnt antagonist DKK1 acts as a tumor suppressor gene that induces apoptosis and inhibits proliferation in human renal cell carcinoma
- (2010) Hiroshi Hirata et al. INTERNATIONAL JOURNAL OF CANCER
- Matrix Metalloproteinase-Induced Epithelial-Mesenchymal Transition in Breast Cancer
- (2010) Evette S. Radisky et al. JOURNAL OF MAMMARY GLAND BIOLOGY AND NEOPLASIA
- Transcriptional regulation of Wnt inhibitory factor-1 by Miz-1/c-Myc
- (2010) J D F Licchesi et al. ONCOGENE
- Blocking transforming growth factor-beta up-regulates E-cadherin and reduces migration and invasion of hepatocellular carcinoma cells
- (2008) Emilia Fransvea et al. HEPATOLOGY
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started