Elevating SOX2 in prostate tumor cells upregulates expression of neuroendocrine genes, but does not reduce the inhibitory effects of enzalutamide
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Title
Elevating SOX2 in prostate tumor cells upregulates expression of neuroendocrine genes, but does not reduce the inhibitory effects of enzalutamide
Authors
Keywords
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Journal
JOURNAL OF CELLULAR PHYSIOLOGY
Volume -, Issue -, Pages -
Publisher
Wiley
Online
2019-10-06
DOI
10.1002/jcp.29267
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Note: Only part of the references are listed.- Sox2 dosage: A critical determinant in the functions of Sox2 in both normal and tumor cells
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- YAP1 regulates prostate cancer stem cell-like characteristics to promote castration resistant growth
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- Modulation of SOX2 expression delineates an end-point for paclitaxel-effectiveness in breast cancer stem cells
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- SOX2 functions as a molecular rheostat to control the growth, tumorigenicity and drug responses of pancreatic ductal adenocarcinoma cells
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- The neuropeptide 26RFa is expressed in human prostate cancer and stimulates the neuroendocrine differentiation and the migration of androgeno-independent prostate cancer cells
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- Elevating SOX2 Levels Deleteriously Affects the Growth of Medulloblastoma and Glioblastoma Cells
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