Dec1 Deficiency Suppresses Cardiac Perivascular Fibrosis Induced by Transverse Aortic Constriction
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Title
Dec1 Deficiency Suppresses Cardiac Perivascular Fibrosis Induced by Transverse Aortic Constriction
Authors
Keywords
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Journal
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
Volume 20, Issue 19, Pages 4967
Publisher
MDPI AG
Online
2019-10-09
DOI
10.3390/ijms20194967
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Note: Only part of the references are listed.- Smad3 suppresses epithelial cell migration and proliferation via the clock gene Dec1, which negatively regulates the expression of clock genes Dec2 and Per1
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- Smad3 and Bmal1 regulate p21 and S100A4 expression in myocardial stromal fibroblasts via TNF-α
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- Disrupting the key circadian regulator CLOCK leads to age-dependent cardiovascular disease
- (2017) Faisal J. Alibhai et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- A Review of the Molecular Mechanisms Underlying the Development and Progression of Cardiac Remodeling
- (2017) Leonardo Schirone et al. Oxidative Medicine and Cellular Longevity
- Global mortality variations in patients with heart failure: results from the International Congestive Heart Failure (INTER-CHF) prospective cohort study
- (2017) Hisham Dokainish et al. Lancet Global Health
- Differentiated embryo chondrocyte 1 (DEC1) is a novel negative regulator of hepatic fibroblast growth factor 21 (FGF21) in aging mice
- (2016) Yu Fujita et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- DEC1 and DEC2 Crosstalk between Circadian Rhythm and Tumor Progression
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- Getting to the Heart of the Matter
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- Tumor Necrosis Factor - Alpha Is Essential for Angiotensin II-Induced Ventricular Remodeling: Role for Oxidative Stress
- (2015) Srinivas Sriramula et al. PLoS One
- S100A4 amplifies TGF-β-induced fibroblast activation in systemic sclerosis
- (2014) Michal Tomcik et al. ANNALS OF THE RHEUMATIC DISEASES
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- Metastasis-associated protein, S100A4 mediates cardiac fibrosis potentially through the modulation of p53 in cardiac fibroblasts
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- The BHLH transcription factor DEC1 plays an important role in the epithelial-mesenchymal transition of pancreatic cancer
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- Pivotal role of cardiomyocyte TGF-β signaling in the murine pathological response to sustained pressure overload
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- Attenuation of myocardial injury in mice with functional deletion of the circadian rhythm gene mPer2
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- Anti-apoptotic effect of the basic helix-loop-helix (bHLH) transcription factor DEC2 in human breast cancer cells
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- Smad3 Mediates Cardiac Inflammation and Fibrosis in Angiotensin II–Induced Hypertensive Cardiac Remodeling
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- Characterization of the inflammatory and fibrotic response in a mouse model of cardiac pressure overload
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