Dapagliflozin rescues endoplasmic reticulum stress-mediated cell death
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Title
Dapagliflozin rescues endoplasmic reticulum stress-mediated cell death
Authors
Keywords
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Journal
Scientific Reports
Volume 9, Issue 1, Pages -
Publisher
Springer Science and Business Media LLC
Online
2019-07-08
DOI
10.1038/s41598-019-46402-6
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- (2018) Sun Ryoung Choi et al. METABOLISM-CLINICAL AND EXPERIMENTAL
- Kcne2 deletion impairs insulin secretion and causes type 2 diabetes mellitus
- (2017) Soo Min Lee et al. FASEB JOURNAL
- Canagliflozin and Cardiovascular and Renal Events in Type 2 Diabetes
- (2017) Bruce Neal et al. NEW ENGLAND JOURNAL OF MEDICINE
- Rtn1a-Mediated Endoplasmic Reticulum Stress in Podocyte Injury and Diabetic Nephropathy
- (2017) Ying Fan et al. Scientific Reports
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- (2016) Ai-Li Cao et al. LABORATORY INVESTIGATION
- Correction: Dapagliflozin, SGLT2 Inhibitor, Attenuates Renal Ischemia-Reperfusion Injury
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- Diabetic Nephropathy: Perspective on Novel Molecular Mechanisms
- (2016) Luigi Gnudi et al. TRENDS IN ENDOCRINOLOGY AND METABOLISM
- Tauroursodeoxycholic Acid Attenuates Renal Tubular Injury in a Mouse Model of Type 2 Diabetes
- (2016) Jing Zhang et al. Nutrients
- Fyn phosphorylates AMPK to inhibit AMPK activity and AMP-dependent activation of autophagy
- (2016) Eijiro Yamada et al. Oncotarget
- Propofol Decreases Endoplasmic Reticulum Stress–Mediated Apoptosis in Retinal Pigment Epithelial Cells
- (2016) Xuezhi Zhou et al. PLoS One
- Empagliflozin, Cardiovascular Outcomes, and Mortality in Type 2 Diabetes
- (2015) Bernard Zinman et al. NEW ENGLAND JOURNAL OF MEDICINE
- Endoplasmic Reticulum Stress in the Diabetic Kidney, the Good, the Bad and the Ugly
- (2015) Robyn Cunard Journal of Clinical Medicine
- Baicalin Ameliorates H2O2 Induced Cytotoxicity in HK-2 Cells through the Inhibition of ER Stress and the Activation of Nrf2 Signaling
- (2014) Miao Lin et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- Long-Term Treatment with the Sodium Glucose Cotransporter 2 Inhibitor, Dapagliflozin, Ameliorates Glucose Homeostasis and Diabetic Nephropathy in db/db Mice
- (2014) Naoto Terami et al. PLoS One
- Clinical Pharmacokinetics and Pharmacodynamics of Dapagliflozin, a Selective Inhibitor of Sodium-Glucose Co-transporter Type 2
- (2013) Sreeneeranj Kasichayanula et al. CLINICAL PHARMACOKINETICS
- Role of ceramide in diabetes mellitus: evidence and mechanisms
- (2013) Sehamuddin Galadari et al. Lipids in Health and Disease
- p58IPK-Mediated Attenuation of the Proapoptotic PERK-CHOP Pathway Allows Malignant Progression upon Low Glucose
- (2013) Anne-Laure Huber et al. MOLECULAR CELL
- Unfolded protein response
- (2012) Stewart Siyan Cao et al. CURRENT BIOLOGY
- Activation of Endoplasmic Reticulum Stress by Hyperglycemia Is Essential for Muller Cell-Derived Inflammatory Cytokine Production in Diabetes
- (2012) Y. Zhong et al. DIABETES
- A novel SGLT is expressed in the human kidney
- (2012) Rajendra K. Kothinti et al. EUROPEAN JOURNAL OF PHARMACOLOGY
- Endothelial Dysfunction in Diabetes Mellitus: Possible Involvement of Endoplasmic Reticulum Stress?
- (2012) Basma Basha et al. Experimental Diabetes Research
- Can We Target Tubular Damage to Prevent Renal Function Decline in Diabetes?
- (2012) Joseph V. Bonventre SEMINARS IN NEPHROLOGY
- Role of Endoplasmic Reticulum Stress in Metabolic Disease and Other Disorders
- (2011) Lale Ozcan et al. Annual Review of Medicine
- Sphingolipids and cancer: ceramide and sphingosine-1-phosphate in the regulation of cell death and drug resistance
- (2010) Suriyan Ponnusamy et al. Future Oncology
- Albumin Overload Induces Apoptosis in Renal Tubular Epithelial Cells through a CHOP-Dependent Pathway
- (2010) Xiaowei Wu et al. OMICS-A JOURNAL OF INTEGRATIVE BIOLOGY
- Dapagliflozin, a Novel SGLT2 Inhibitor, Induces Dose-Dependent Glucosuria in Healthy Subjects
- (2009) B Komoroski et al. CLINICAL PHARMACOLOGY & THERAPEUTICS
- Dapagliflozin, a Novel, Selective SGLT2 Inhibitor, Improved Glycemic Control Over 2 Weeks in Patients With Type 2 Diabetes Mellitus
- (2009) B Komoroski et al. CLINICAL PHARMACOLOGY & THERAPEUTICS
- Renal sodium–glucose transport: role in diabetes mellitus and potential clinical implications
- (2009) George L. Bakris et al. KIDNEY INTERNATIONAL
- Apoptosis induced by endoplasmic reticulum stress involved in diabetic kidney disease
- (2008) Guanghui Liu et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Plasma Ceramides Are Elevated in Obese Subjects With Type 2 Diabetes and Correlate With the Severity of Insulin Resistance
- (2008) J. M. Haus et al. DIABETES
- From endoplasmic-reticulum stress to the inflammatory response
- (2008) Kezhong Zhang et al. NATURE
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