The tumor cell‐secreted matricellular protein WISP 1 drives pro‐metastatic collagen linearization
Published 2019 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
The tumor cell‐secreted matricellular protein
WISP
1 drives pro‐metastatic collagen linearization
Authors
Keywords
-
Journal
EMBO JOURNAL
Volume -, Issue -, Pages -
Publisher
EMBO
Online
2019-07-11
DOI
10.15252/embj.2018101302
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Tumor elastography and its association with collagen and the tumor microenvironment.
- (2018) Johannes Riegler et al. CLINICAL CANCER RESEARCH
- Sup35NMp morphology evaluation on Au, Si, formvar and mica surfaces using AFM, SEM and TEM
- (2018) P.A. Sokolov et al. JOURNAL OF STRUCTURAL BIOLOGY
- CCN4/WISP1 controls cutaneous wound healing by modulating proliferation, migration and ECM expression in dermal fibroblasts via α5β1 and TNFα
- (2018) Mitsuaki Ono et al. MATRIX BIOLOGY
- A tension-mediated glycocalyx–integrin feedback loop promotes mesenchymal-like glioblastoma
- (2018) J. Matthew Barnes et al. NATURE CELL BIOLOGY
- Mechanoreciprocity in cell migration
- (2017) Sjoerd van Helvert et al. NATURE CELL BIOLOGY
- Tumor Cell-Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
- (2016) M. J. Oudin et al. Cancer Discovery
- High expression of WISP1 in colon cancer is associated with apoptosis, invasion and poor prognosis
- (2016) Jianghong Wu et al. Oncotarget
- Identification and validation ofWISP1as an epigenetic regulator of metastasis in oral squamous cell carcinoma
- (2015) Martijn J. A. M. Clausen et al. GENES CHROMOSOMES & CANCER
- Human breast cancer invasion and aggression correlates with ECM stiffening and immune cell infiltration
- (2015) I. Acerbi et al. Integrative Biology
- CCN4/WISP1 (WNT1 inducible signaling pathway protein 1): A focus on its role in cancer
- (2015) Irem Gurbuz et al. INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY
- WNT1-induced Secreted Protein-1 (WISP1), a Novel Regulator of Bone Turnover and Wnt Signaling
- (2015) Azusa Maeda et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- WNT-1 inducible signaling pathway protein-1 enhances growth and tumorigenesis in human breast cancer
- (2015) Kun-Chun Chiang et al. Scientific Reports
- Targeting ECM Disrupts Cancer Progression
- (2015) Freja A. Venning et al. Frontiers in Oncology
- Fibrin binds to collagen and provides a bridge for αVβ3 integrin-dependent contraction of collagen gels
- (2014) Vahid Reyhani et al. BIOCHEMICAL JOURNAL
- Collagen I Self-Assembly: Revealing the Developing Structures that Generate Turbidity
- (2014) Jieling Zhu et al. BIOPHYSICAL JOURNAL
- 3D Collagen Alignment Limits Protrusions to Enhance Breast Cancer Cell Persistence
- (2014) Kristin M. Riching et al. BIOPHYSICAL JOURNAL
- Quantitative and unbiased analysis of directional persistence in cell migration
- (2014) Roman Gorelik et al. Nature Protocols
- Extracellular matrix signatures of human mammary carcinoma identify novel metastasis promoters
- (2014) Alexandra Naba et al. eLife
- voom: precision weights unlock linear model analysis tools for RNA-seq read counts
- (2014) Charity W Law et al. GENOME BIOLOGY
- The secretome in cancer progression
- (2013) James L. Paltridge et al. BIOCHIMICA ET BIOPHYSICA ACTA-PROTEINS AND PROTEOMICS
- Microfabricated collagen tracks facilitate single cell metastatic invasion in 3D
- (2013) Casey M. Kraning-Rush et al. Integrative Biology
- Physical limits of cell migration: Control by ECM space and nuclear deformation and tuning by proteolysis and traction force
- (2013) Katarina Wolf et al. JOURNAL OF CELL BIOLOGY
- Microenvironmental regulation of tumor progression and metastasis
- (2013) Daniela F Quail et al. NATURE MEDICINE
- STAR: ultrafast universal RNA-seq aligner
- (2012) Alexander Dobin et al. BIOINFORMATICS
- The extracellular matrix: A dynamic niche in cancer progression
- (2012) Pengfei Lu et al. JOURNAL OF CELL BIOLOGY
- Molecular Characteristics and Metastasis Predictor Genes of Triple-Negative Breast Cancer: A Clinical Study of Triple-Negative Breast Carcinomas
- (2012) Wen-Hung Kuo et al. PLoS One
- Aligned Collagen Is a Prognostic Signature for Survival in Human Breast Carcinoma
- (2011) Matthew W. Conklin et al. AMERICAN JOURNAL OF PATHOLOGY
- Molecular signatures database (MSigDB) 3.0
- (2011) A. Liberzon et al. BIOINFORMATICS
- TGF-β signaling in fibrosis
- (2011) Anna Biernacka et al. GROWTH FACTORS
- In situ force mapping of mammary gland transformation
- (2011) Jose I. Lopez et al. Integrative Biology
- A Genomic Predictor of Response and Survival Following Taxane-Anthracycline Chemotherapy for Invasive Breast Cancer
- (2011) Christos Hatzis JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION
- The Matrisome:In SilicoDefinition andIn VivoCharacterization by Proteomics of Normal and Tumor Extracellular Matrices
- (2011) Alexandra Naba et al. MOLECULAR & CELLULAR PROTEOMICS
- Siah Regulation of Pard3A Controls Neuronal Cell Adhesion During Germinal Zone Exit
- (2010) J. K. Famulski et al. SCIENCE
- An online survival analysis tool to rapidly assess the effect of 22,277 genes on breast cancer prognosis using microarray data of 1,809 patients
- (2009) Balazs Györffy et al. BREAST CANCER RESEARCH AND TREATMENT
- Matrix Crosslinking Forces Tumor Progression by Enhancing Integrin Signaling
- (2009) Kandice R. Levental et al. CELL
- Vascular Endothelial Cadherin Promotes Breast Cancer Progression via Transforming Growth Factor Signaling
- (2008) M. Labelle et al. CANCER RESEARCH
- TGFβ in Cancer
- (2008) Joan Massagué CELL
- Functions and mechanisms of action of CCN matricellular proteins
- (2008) Chih-Chiun Chen et al. INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY
- Cellular and molecular mechanisms of fibrosis
- (2007) TA Wynn JOURNAL OF PATHOLOGY
Create your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create NowAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started