Curcumin Modulates Paraquat-Induced Epithelial to Mesenchymal Transition by Regulating Transforming Growth Factor-β (TGF-β) in A549 Cells
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Title
Curcumin Modulates Paraquat-Induced Epithelial to Mesenchymal Transition by Regulating Transforming Growth Factor-β (TGF-β) in A549 Cells
Authors
Keywords
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Journal
INFLAMMATION
Volume -, Issue -, Pages -
Publisher
Springer Science and Business Media LLC
Online
2019-04-26
DOI
10.1007/s10753-019-01006-0
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Note: Only part of the references are listed.- Curcumin Suppresses Intestinal Fibrosis by Inhibition of PPARγ-Mediated Epithelial-Mesenchymal Transition
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- Curcumin inhibits paraquat induced lung inflammation and fibrosis by extracellular matrix modifications in mouse model
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- The innate immune function of airway epithelial cells in inflammatory lung disease
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- Involvement of epithelial-to-mesenchymal transition and associated transforming growth factor-β/Smad signaling in paraquat-induced pulmonary fibrosis
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- Paraquat Induces Epithelial-Mesenchymal Transition-Like Cellular Response Resulting in Fibrogenesis and the Prevention of Apoptosis in Human Pulmonary Epithelial Cells
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- Alveolar Epithelial Cells Are Critical in Protection of the Respiratory Tract by Secretion of Factors Able To Modulate the Activity of Pulmonary Macrophages and Directly Control Bacterial Growth
- (2012) Olga D. Chuquimia et al. INFECTION AND IMMUNITY
- Innate Immunity in the Respiratory Epithelium
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- The extrinsic and intrinsic apoptotic pathways are involved in manganese toxicity in rat astrocytoma C6 cells
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- Contribution of Epithelial-derived Fibroblasts to Bleomycin-induced Lung Fibrosis
- (2009) Harikrishna Tanjore et al. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE
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- Cell mechanics of alveolar epithelial cells (AECs) and macrophages (AMs)
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- Paraquat Poisonings: Mechanisms of Lung Toxicity, Clinical Features, and Treatment
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