Pancreatic β-cell function is inhibited by miR-3666 in type 2 diabetes mellitus by targeting adiponectin
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Title
Pancreatic β-cell function is inhibited by miR-3666 in type 2 diabetes mellitus by targeting adiponectin
Authors
Keywords
-
Journal
BRAZILIAN JOURNAL OF MEDICAL AND BIOLOGICAL RESEARCH
Volume 52, Issue 6, Pages -
Publisher
FapUNIFESP (SciELO)
Online
2019-05-26
DOI
10.1590/1414-431x20198344
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Related references
Note: Only part of the references are listed.- miR-20b, miR-296, and Let-7f Expression in Human Adipose Tissue is Related to Obesity and Type 2 Diabetes
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- Emerging roles of microRNAs in cancer
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- The role of microRNAs in islet β-cell development
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- MicroRNA-3666 Regulates Thyroid Carcinoma Cell Proliferation via MET
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- MicroRNA-19a-3p enhances the proliferation and insulin secretion, while it inhibits the apoptosis of pancreatic β cells via the inhibition of SOCS3
- (2016) Yuan Li et al. INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE
- Role of miRNAs in the pathogenesis and susceptibility of diabetes mellitus
- (2016) Naoko Hashimoto et al. JOURNAL OF HUMAN GENETICS
- MicroRNA-3666-induced suppression of SIRT7 inhibits the growth of non-small cell lung cancer cells
- (2016) Hongyang Shi et al. ONCOLOGY REPORTS
- The microRNA-200 family regulates pancreatic beta cell survival in type 2 diabetes
- (2015) Bengt-Frederik Belgardt et al. NATURE MEDICINE
- MicroRNA 144 Impairs Insulin Signaling by Inhibiting the Expression of Insulin Receptor Substrate 1 in Type 2 Diabetes Mellitus
- (2011) Dwi Setyowati Karolina et al. PLoS One
- Control of glucose homeostasis and insulin sensitivity by the Let-7 family of microRNAs
- (2011) R. J. A. Frost et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Circulating serum adiponectin levels in patients with coronary artery disease: relationship to atherosclerotic burden and cardiac function
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