Studies on mechanism of free Nε‐(carboxymethyl)lysine‐induced toxic injury in mice
Published 2019 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Studies on mechanism of free Nε‐(carboxymethyl)lysine‐induced toxic injury in mice
Authors
Keywords
-
Journal
JOURNAL OF BIOCHEMICAL AND MOLECULAR TOXICOLOGY
Volume -, Issue -, Pages e22322
Publisher
Wiley
Online
2019-03-29
DOI
10.1002/jbt.22322
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Accumulation of the advanced glycation end product carboxymethyl lysine in breast cancer is positively associated with estrogen receptor expression and unfavorable prognosis in estrogen receptor-negative cases
- (2016) Norbert Nass et al. HISTOCHEMISTRY AND CELL BIOLOGY
- Toxicological evaluation of advanced glycation end product Nε-(carboxymethyl)lysine: Acute and subacute oral toxicity studies
- (2016) Xin Liu et al. REGULATORY TOXICOLOGY AND PHARMACOLOGY
- Dietary CML-enriched protein induces functional arterial aging in a RAGE-dependent manner in mice
- (2015) Nicolas Grossin et al. MOLECULAR NUTRITION & FOOD RESEARCH
- Acute secondhand smoke-induced pulmonary inflammation is diminished in RAGE knockout mice
- (2014) Tyler T. Wood et al. AMERICAN JOURNAL OF PHYSIOLOGY-LUNG CELLULAR AND MOLECULAR PHYSIOLOGY
- Nε-carboxymethyllysine-mediated endoplasmic reticulum stress promotes endothelial cell injury through Nox4/MKP-3 interaction
- (2014) Wen-Jane Lee et al. FREE RADICAL BIOLOGY AND MEDICINE
- Effects of Nε-carboxymethyl-Lysine on ERS-mediated apoptosis in diabetic atherosclerosis
- (2014) Zhongqun Wang et al. INTERNATIONAL JOURNAL OF CARDIOLOGY
- Advanced glycation endproducts in food and their effects on health
- (2013) Malene W. Poulsen et al. FOOD AND CHEMICAL TOXICOLOGY
- Digestibility of extruded proteins and metabolic transit of N ε -carboxymethyllysine in rats
- (2012) Issam Alamir et al. AMINO ACIDS
- Advanced glycation end product Nε-carboxymethyllysine induces endothelial cell injury: the involvement of SHP-1-regulated VEGFR-2 dephosphorylation
- (2012) Shing Hwa Liu et al. JOURNAL OF PATHOLOGY
- Advanced Glycation End Products (AGEs) Induce Apoptosis via a Novel Pathway
- (2011) Sidharth Mahali et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- A diet based on high-heat-treated foods promotes risk factors for diabetes mellitus and cardiovascular diseases
- (2010) Inès Birlouez-Aragon et al. AMERICAN JOURNAL OF CLINICAL NUTRITION
- Does Accumulation of Advanced Glycation End Products Contribute to the Aging Phenotype?
- (2010) R. D. Semba et al. JOURNALS OF GERONTOLOGY SERIES A-BIOLOGICAL SCIENCES AND MEDICAL SCIENCES
- Rapid glycation with D-ribose induces globular amyloid-like aggregations of BSA with high cytotoxicity to SH-SY5Y cells
- (2009) Yan Wei et al. BMC CELL BIOLOGY
- Advanced glycation endproducts and their receptor RAGE in Alzheimer's disease
- (2009) Velandai Srikanth et al. NEUROBIOLOGY OF AGING
- Possible link of food-derived advanced glycation end products (AGEs) to the development of diabetes
- (2008) S. Yamagishi et al. MEDICAL HYPOTHESES
- Nɛ-carboxymethyllysine-modified proteins are unable to bind to RAGE and activate an inflammatory response
- (2008) Timo M. Buetler et al. MOLECULAR NUTRITION & FOOD RESEARCH
Create your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create NowAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started