Multifunctional ion transport properties of human SLC4A11: comparison of the SLC4A11-B and SLC4A11-C variants

Transcriptomic Analysis of Cultured Corneal Endothelial Cells as a Validation for Their Use in Cell Replacement Therapy

(2016) Ricardo F. Frausto et al.   CELL TRANSPLANTATION

Human SLC4A11-C functions as a DIDS-stimulatable H+(OH−) permeation pathway: partial correction of R109H mutant transport

(2015) Liyo Kao et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Mutations in the Corneal Endothelial Dystrophy–Associated Gene SLC4A11 Render the Cells More Vulnerable to Oxidative Insults

(2015) Sanhita Roy et al.   CORNEA

Human SLC4A11 Is a Novel NH3/H+Co-transporter

(2015) Wenlin Zhang et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Whole exome sequence analysis of Peters anomaly

(2014) Eric Weh et al.   HUMAN GENETICS

Biosynthetic and functional defects in newly identified SLC4A11 mutants and absence of COL8A2 mutations in Fuchs endothelial corneal dystrophy

(2014) Nagasamy Soumittra et al.   JOURNAL OF HUMAN GENETICS

Interactions between the N-terminal tail and the gating machinery of hERG K+ channels both in closed and open/inactive states

(2014) Pilar de la Peña et al.   PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY

Ion channels and apoptosis in cancer

(2014) C. D. Bortner et al.   PHILOSOPHICAL TRANSACTIONS OF THE ROYAL SOCIETY B-BIOLOGICAL SCIENCES

SLC4A11 is an EIPA-sensitive Na+ permeable pHi regulator

(2013) Diego G. Ogando et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Transmembrane water-flux through SLC4A11: a route defective in genetic corneal diseases

(2013) Gonzalo L. Vilas et al.   HUMAN MOLECULAR GENETICS

Knockdown a Water Channel Protein, Aquaporin-4, Induced Glioblastoma Cell Apoptosis

(2013) Ting Ding et al.   PLoS One

Primer-BLAST: A tool to design target-specific primers for polymerase chain reaction

(2012) Jian Ye et al.   BMC BIOINFORMATICS

Cytoplasmic Domains and Voltage-Dependent Potassium Channel Gating

(2012) Francisco Barros et al.   Frontiers in Pharmacology

Oligomerization of SLC4A11 protein and the severity of FECD and CHED2 corneal dystrophies caused bySLC4A11mutations

(2011) Gonzalo L. Vilas et al.   HUMAN MUTATION

Evidence of Oxidative Stress in the Pathogenesis of Fuchs Endothelial Corneal Dystrophy

(2010) Ula V. Jurkunas et al.   AMERICAN JOURNAL OF PATHOLOGY

Missense mutations in the sodium borate cotransporter SLC4A11 cause late-onset Fuchs corneal dystrophya

(2010) S. Amer Riazuddin et al.   HUMAN MUTATION

SLC4A11 Prevents Osmotic Imbalance Leading to Corneal Endothelial Dystrophy, Deafness, and Polyuria

(2010) Nicole Gröger et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Slc4a11Gene Disruption in Mice

(2009) Ivan A. Lopez et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Channel-like slippage modes in the human anion/proton exchanger ClC-4

(2009) Alexi K. Alekov et al.   JOURNAL OF GENERAL PHYSIOLOGY

Water-Transporting Proteins

(2009) Thomas Zeuthen   JOURNAL OF MEMBRANE BIOLOGY

Identification of Mutations in the SLC4A11 Gene in Patients With Recessive Congenital Hereditary Endothelial Dystrophy

(2008) Boomiraj Hemadevi   ARCHIVES OF OPHTHALMOLOGY