Emodin ameliorates cisplatin-induced apoptosis of rat renal tubular cells in vitro by activating autophagy

Autophagy protects renal tubular cells against cyclosporine toxicity

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Autophagy delays apoptosis in renal tubular epithelial cells in cisplatin cytotoxicity

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Emodin Regulates Glucose Utilization by Activating AMP-activated Protein Kinase

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Interleukin-6 modulates oxidative stress produced during the development of cisplatin nephrotoxicity

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Intervention in Genotoxic Stress–Induced Senescence by Cordycepin Through Activation of eIF2α and Suppression of Sp1

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Aloe-emodin suppresses prostate cancer by targeting the mTOR complex 2

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Protective effects of emodin against cisplatin-induced oxidative stress in cultured human kidney (HEK 293) cells

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ER signaling regulation drives the switch between autophagy and apoptosis in NRK-52E cells exposed to cisplatin

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Abrogation of cisplatin-induced nephrotoxicity by emodin in rats

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Sphingosine Kinase-1 Pathway Mediates High Glucose-Induced Fibronectin Expression in Glomerular Mesangial Cells

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Emodin and [6]-gingerol lessen hypoxia-induced embryotoxicities in cultured mouse whole embryos via upregulation of hypoxia-inducible factor 1α and intracellular superoxide dismutases

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LKB1 and AMP-activated protein kinase control of mTOR signalling and growth

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Emodin ameliorates high-glucose induced mesangial p38 over-activation and hypocontractility via activation of PPARγ

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Emodin suppresses cell proliferation and fibronectin expression via p38MAPK pathway in rat mesangial cells cultured under high glucose

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Chemotherapy-associated renal dysfunction

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Autophagy is cytoprotective during cisplatin injury of renal proximal tubular cells

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Autophagy fights disease through cellular self-digestion

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