Interleukin-33 promotes inflammatory cytokine production in chronic airway inflammation
Published 2015 View Full Article
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Title
Interleukin-33 promotes inflammatory cytokine production in chronic airway inflammation
Authors
Keywords
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Journal
Biochemistry and Cell Biology
Volume 93, Issue 4, Pages 359-366
Publisher
Canadian Science Publishing
Online
2015-04-22
DOI
10.1139/bcb-2014-0163
References
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Related references
Note: Only part of the references are listed.- Increased IL-33 expression in chronic obstructive pulmonary disease
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- Cigarette smoke alters IL-33 expression and release in airway epithelial cells
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- Modulation of IL-8 Boosted by Mycoplasma pneumoniae lysate in Human Airway Epithelial Cells
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- Long-term IL-33–producing epithelial progenitor cells in chronic obstructive lung disease
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- The paracrine effect of cancer-associated fibroblast-induced interleukin-33 regulates the invasiveness of head and neck squamous cell carcinoma
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- Effect of lipoteichoic acid on IL-2 and IL-5 release from T lymphocytes in asthma and COPD
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- Histone deacetylase activity is decreased in peripheral blood monocytes in patients with COPD
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- Inflammatory Bowel Disease-Associated Interleukin-33 Is Preferentially Expressed in Ulceration-Associated Myofibroblasts
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- Reduced heat shock protein 70 in airway smooth muscle in patients with chronic obstructive pulmonary disease
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- IL-33 amplifies both Th1- and Th2-type responses through its activity on human basophils, allergen-reactive Th2 cells, iNKT and NK Cells
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- A novel IL-1 family cytokine, IL-33, potently activates human eosinophils
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