Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

Published 2018 View Full Article

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Title
Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation
Authors
Keywords
autophagy, innate immunity, complement system, C3, ATG16L1, beta cell, type 2 diabetes
Journal
Cell Metabolism
Volume 29, Issue 1, Pages 202-210.e6
Publisher
Elsevier BV
Online
2018-10-04
DOI
10.1016/j.cmet.2018.09.009

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