Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

标题
Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation
作者
关键词
autophagy, innate immunity, complement system, C3, ATG16L1, beta cell, type 2 diabetes
出版物
Cell Metabolism
Volume 29, Issue 1, Pages 202-210.e6
出版商
Elsevier BV
发表日期
2018-10-04
DOI
10.1016/j.cmet.2018.09.009

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