The ER stress inducer DMC enhances TRAIL-induced apoptosis in glioblastoma
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Title
The ER stress inducer DMC enhances TRAIL-induced apoptosis in glioblastoma
Authors
Keywords
TNF-related apoptosis-inducing ligand (TRAIL), Glioblastoma multiforme (GBM), 2,5-dimethyl-celecoxib (DMC), Endoplasmic reticulum (ER) stress, Apoptosis
Journal
SpringerPlus
Volume 3, Issue 1, Pages 495
Publisher
Springer Nature
Online
2014-09-01
DOI
10.1186/2193-1801-3-495
References
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- Rapid and efficient cancer cell killing mediated by high-affinity death receptor homotrimerizing TRAIL variants
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- Stem-cell-like glioma cells are resistant to TRAIL/Apo2L and exhibit down-regulation of caspase-8 by promoter methylation
- (2009) David Capper et al. ACTA NEUROPATHOLOGICA
- Aggravated Endoplasmic Reticulum Stress as a Basis for Enhanced Glioblastoma Cell Killing by Bortezomib in Combination with Celecoxib or Its Non-Coxib Analogue, 2,5-Dimethyl-Celecoxib
- (2008) A. Kardosh et al. CANCER RESEARCH
- Shiga toxin 1 induces apoptosis through the endoplasmic reticulum stress response in human monocytic cells
- (2008) Sang-Yun Lee et al. CELLULAR MICROBIOLOGY
- TRAIL-mediated apoptosis in malignant glioma cells is augmented by celecoxib through proteasomal degradation of survivin
- (2008) T. Gaiser et al. NEUROSCIENCE LETTERS
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