Genome Instability at Common Fragile Sites: Searching for the Cause of Their Instability
Published 2013 View Full Article
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Title
Genome Instability at Common Fragile Sites: Searching for the Cause of Their Instability
Authors
Keywords
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Journal
Biomed Research International
Volume 2013, Issue -, Pages 1-9
Publisher
Hindawi Limited
Online
2013-09-06
DOI
10.1155/2013/730714
References
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Related references
Note: Only part of the references are listed.- Perturbed replication induced genome wide or at common fragile sites is differently managed in the absence of WRN
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- Distinct Roles of Mus81, Yen1, Slx1-Slx4, and Rad1 Nucleases in the Repair of Replication-Born Double-Strand Breaks by Sister Chromatid Exchange
- (2012) S. Munoz-Galvan et al. MOLECULAR AND CELLULAR BIOLOGY
- R Loops: From Transcription Byproducts to Threats to Genome Stability
- (2012) Andrés Aguilera et al. MOLECULAR CELL
- The WRN and MUS81 proteins limit cell death and genome instability following oncogene activation
- (2012) I Murfuni et al. ONCOGENE
- Collisions between Replication and Transcription Complexes Cause Common Fragile Site Instability at the Longest Human Genes
- (2011) Anne Helmrich et al. MOLECULAR CELL
- Failure of Origin Activation in Response to Fork Stalling Leads to Chromosomal Instability at Fragile Sites
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- Cell-type-specific replication initiation programs set fragility of the FRA3B fragile site
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- Molecular profiling of common fragile sites in human fibroblasts
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- Fanconi anemia and Bloom's syndrome crosstalk through FANCJ–BLM helicase interaction
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- Common fragile sites: mechanisms of instability revisited
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- Roles of Werner syndrome protein in protection of genome integrity
- (2010) Marie L. Rossi et al. DNA REPAIR
- ATR and ATM differently regulate WRN to prevent DSBs at stalled replication forks and promote replication fork recovery
- (2010) Francesca Ammazzalorso et al. EMBO JOURNAL
- ATR preferentially interacts with common fragile site FRA3B and the binding requires its kinase activity in response to aphidicolin treatment
- (2010) Cheng Wan et al. MUTATION RESEARCH-FUNDAMENTAL AND MOLECULAR MECHANISMS OF MUTAGENESIS
- Maintaining genome stability at the replication fork
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- Genomic instability — an evolving hallmark of cancer
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- Rad51 protects nascent DNA from Mre11-dependent degradation and promotes continuous DNA synthesis
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- The checkpoint response to replication stress
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- Claspin inhibition leads to fragile site expression
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- Replication stress induces sister-chromatid bridging at fragile site loci in mitosis
- (2009) Kok Lung Chan et al. NATURE CELL BIOLOGY
- The FANC pathway and BLM collaborate during mitosis to prevent micro-nucleation and chromosome abnormalities
- (2009) Valeria Naim et al. NATURE CELL BIOLOGY
- DNA structure and the Werner protein modulate human DNA polymerase delta-dependent replication dynamics within the common fragile site FRA16D
- (2009) Sandeep N. Shah et al. NUCLEIC ACIDS RESEARCH
- Mechanisms of Dealing with DNA Damage-Induced Replication Problems
- (2008) Magda Budzowska et al. CELL BIOCHEMISTRY AND BIOPHYSICS
- Replication timing of two human common fragile sites: FRA1H and FRA2G
- (2008) F. Pelliccia et al. CYTOGENETIC AND GENOME RESEARCH
- Stably transfected common fragile site sequences exhibit instability at ectopic sites
- (2008) Ryan L. Ragland et al. GENES CHROMOSOMES & CANCER
- Werner syndrome helicase activity is essential in maintaining fragile site stability
- (2008) Livia Maria Pirzio et al. JOURNAL OF CELL BIOLOGY
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- (2008) Kerstin Gari et al. MOLECULAR CELL
- Human chromosome fragility
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