PINK1 deficiency in  -cells increases basal insulin secretion and improves glucose tolerance in mice

Predicting Risk of Type 2 Diabetes Mellitus with Genetic Risk Models on the Basis of Established Genome-wide Association Markers: A Systematic Review

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Nkx6.1 Is Essential for Maintaining the Functional State of Pancreatic Beta Cells

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Risk of Parkinson Disease Onset in Patients With Diabetes: A 9-year population-based cohort study with age and sex stratifications

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Cell metabolism affects selective vulnerability in PINK1-associated Parkinson's disease

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Clinical features of Parkinson disease when onset of diabetes came first: A case-control study

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Role for Insulin Signaling in Catecholaminergic Neurons in Control of Energy Homeostasis

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Insulin resistance impairs nigrostriatal dopamine function

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Variation in the PTEN-induced putative kinase 1 gene associated with the increase risk of type 2 diabetes in northern Chinese

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Pancreatic β Cells Require NeuroD to Achieve and Maintain Functional Maturity

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PGC-1 , A Potential Therapeutic Target for Early Intervention in Parkinson's Disease

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Mitophagy: the latest problem for Parkinson's disease

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From the Triumvirate to the Ominous Octet: A New Paradigm for the Treatment of Type 2 Diabetes Mellitus

(2009) R. A. DeFronzo   DIABETES

PINK1 function in health and disease

(2009) Emma Deas et al.   EMBO Molecular Medicine

Mutant Pink1 induces mitochondrial dysfunction in a neuronal cell model of Parkinson’s disease by disturbing calcium flux

(2009) Roberta Marongiu et al.   JOURNAL OF NEUROCHEMISTRY

PINK1-Associated Parkinson's Disease Is Caused by Neuronal Vulnerability to Calcium-Induced Cell Death

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Down-Regulation of Hepatic HNF4α Gene Expression during Hyperinsulinemia via SREBPs

(2009) Xuefen Xie et al.   MOLECULAR ENDOCRINOLOGY

A protocol for islet isolation from mouse pancreas

(2009) Dong-Sheng Li et al.   Nature Protocols

PINK1 Is Necessary for Long Term Survival and Mitochondrial Function in Human Dopaminergic Neurons

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