Histone deacetylases 1 and 2 maintain S-phase chromatin and DNA replication fork progression
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Title
Histone deacetylases 1 and 2 maintain S-phase chromatin and DNA replication fork progression
Authors
Keywords
Hdacs1, Hdacs2, Replication, Nascent chromatin, Chromatin remodelers
Journal
Epigenetics & Chromatin
Volume 6, Issue 1, Pages 27
Publisher
Springer Nature
Online
2013-08-16
DOI
10.1186/1756-8935-6-27
References
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- (2012) Andrew N. Blackford et al. HUMAN MOLECULAR GENETICS
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- (2012) Geneviève P Delcuve et al. Clinical Epigenetics
- SnapShot: Chromatin Remodeling: ISWI
- (2011) Adam N. Yadon et al. CELL
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- (2011) B. M. Sirbu et al. GENES & DEVELOPMENT
- Proliferating cell nuclear antigen (PCNA): a key factor in DNA replication and cell cycle regulation
- (2010) Wojciech Strzalka et al. ANNALS OF BOTANY
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- (2010) Chiara Conti et al. CANCER RESEARCH
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- (2010) Roel H Wilting et al. EMBO JOURNAL
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- (2010) Kyle M Miller et al. NATURE STRUCTURAL & MOLECULAR BIOLOGY
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- (2009) R. Driscoll et al. GENES & DEVELOPMENT
- Genetic dissection of histone deacetylase requirement in tumor cells
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- (2008) C. James Chou et al. JOURNAL OF BIOLOGICAL CHEMISTRY
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- (2008) Karlene A. Cimprich et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
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