The inactivation of RNase G reduces the Stenotrophomonas maltophilia susceptibility to quinolones by triggering the heat shock response
Published 2015 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
The inactivation of RNase G reduces the Stenotrophomonas maltophilia susceptibility to quinolones by triggering the heat shock response
Authors
Keywords
-
Journal
Frontiers in Microbiology
Volume 6, Issue -, Pages -
Publisher
Frontiers Media SA
Online
2015-10-19
DOI
10.3389/fmicb.2015.01068
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- High-level quinolone resistance is associated with the overexpression of smeVWX in Stenotrophomonas maltophilia clinical isolates
- (2015) G. García-León et al. CLINICAL MICROBIOLOGY AND INFECTION
- A Function of SmeDEF, the Major Quinolone Resistance Determinant of Stenotrophomonas maltophilia, Is the Colonization of Plant Roots
- (2014) Guillermo García-León et al. APPLIED AND ENVIRONMENTAL MICROBIOLOGY
- Interplay between intrinsic and acquired resistance to quinolones inStenotrophomonas maltophilia
- (2014) Guillermo García-León et al. ENVIRONMENTAL MICROBIOLOGY
- Biofilm Formation and Antibiotic Resistance in Salmonella Typhimurium Are Affected by Different Ribonucleases
- (2014) Margarida Saramago et al. JOURNAL OF MICROBIOLOGY AND BIOTECHNOLOGY
- What is a resistance gene? Ranking risk in resistomes
- (2014) José L. Martínez et al. NATURE REVIEWS MICROBIOLOGY
- Stenotrophomonas maltophilia: an Emerging Global Opportunistic Pathogen
- (2012) J. S. Brooke CLINICAL MICROBIOLOGY REVIEWS
- Overproduction of the multidrug efflux pump MexEF-OprN does not impair Pseudomonas aeruginosa fitness in competition tests, but produces specific changes in bacterial regulatory networks
- (2012) Jorge Olivares et al. ENVIRONMENTAL MICROBIOLOGY
- Whole-Genome Sequence of Stenotrophomonas maltophilia D457, a Clinical Isolate and a Model Strain
- (2012) F. Lira et al. JOURNAL OF BACTERIOLOGY
- Involvement of an ATP-Dependent Protease, PA0779/AsrA, in Inducing Heat Shock in Response to Tobramycin in Pseudomonas aeruginosa
- (2011) Kristen N. Kindrachuk et al. ANTIMICROBIAL AGENTS AND CHEMOTHERAPY
- Quinolone Resistance: Much More than Predicted
- (2011) Alvaro Hernández et al. Frontiers in Microbiology
- DnaK and GroEL are induced in response to antibiotic and heat shock in Acinetobacter baumannii
- (2010) K. Cardoso et al. JOURNAL OF MEDICAL MICROBIOLOGY
- SmQnr Contributes to Intrinsic Resistance to Quinolones in Stenotrophomonas maltophilia
- (2009) M. B. Sanchez et al. ANTIMICROBIAL AGENTS AND CHEMOTHERAPY
- Molecular cloning and characterization of SmrA, a novel ABC multidrug efflux pump from Stenotrophomonas maltophilia
- (2009) A. Al-Hamad et al. JOURNAL OF ANTIMICROBIAL CHEMOTHERAPY
- Activity of Levofloxacin Alone and in Combination with a DnaK Inhibitor against Gram-Negative Rods, Including Levofloxacin-Resistant Strains
- (2008) K. Credito et al. ANTIMICROBIAL AGENTS AND CHEMOTHERAPY
- Smqnr, a New Chromosome-Carried Quinolone Resistance Gene in Stenotrophomonas maltophilia
- (2008) K. Shimizu et al. ANTIMICROBIAL AGENTS AND CHEMOTHERAPY
- Predictive analysis of transmissible quinolone resistance indicates Stenotrophomonas maltophilia as a potential source of a novel family of Qnr determinants
- (2008) María B Sánchez et al. BMC MICROBIOLOGY
- Mapping and quantifying mammalian transcriptomes by RNA-Seq
- (2008) Ali Mortazavi et al. NATURE METHODS
- The Neglected Intrinsic Resistome of Bacterial Pathogens
- (2008) Alicia Fajardo et al. PLoS One
- The complete genome, comparative and functional analysis of Stenotrophomonas maltophilia reveals an organism heavily shielded by drug resistance determinants
- (2008) Lisa C Crossman et al. GENOME BIOLOGY
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started