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Title
Dioxin Inhibits Zebrafish Epicardium and Proepicardium Development
Authors
Keywords
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Journal
TOXICOLOGICAL SCIENCES
Volume 131, Issue 2, Pages 558-567
Publisher
Oxford University Press (OUP)
Online
2012-11-08
DOI
10.1093/toxsci/kfs301
References
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Related references
Note: Only part of the references are listed.- AHR2 Mutant Reveals Functional Diversity of Aryl Hydrocarbon Receptors in Zebrafish
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- tcf21+ epicardial cells adopt non-myocardial fates during zebrafish heart development and regeneration
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- Tbx5 and Bmp Signaling Are Essential for Proepicardium Specification in Zebrafish
- (2010) Jiandong Liu et al. CIRCULATION RESEARCH
- Epicardial–Myocardial Signaling Directing Coronary Vasculogenesis
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- Zebrafish cardiac enhancer trap lines: New tools for in vivo studies of cardiovascular development and disease
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- The embryonic epicardium: an essential element of cardiac development
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- Perspectives on the Potential Involvement of the Ah Receptor-Dioxin Axis in Cardiovascular Disease
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- Epicardial cells are missing from the surface of hearts with ischemic cardiomyopathy: A useful clue about the self-renewal potential of the adult human heart?
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- Persistent Adverse Effects on Health and Reproduction Caused by Exposure of Zebrafish to 2,3,7,8-Tetrachlorodibenzo-p-dioxin During Early Development and Gonad Differentiation
- (2009) Tisha C. King Heiden et al. TOXICOLOGICAL SCIENCES
- Embryonic development of the proepicardium and coronary vessels
- (2008) Anna Ratajska et al. INTERNATIONAL JOURNAL OF DEVELOPMENTAL BIOLOGY
- PCB126 Exposure Disrupts ZebraFish Ventricular and Branchial but Not Early Neural Crest Development
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- 2,3,7,8-Tetrachlorodibenzo-p-dioxin Exposure Prevents Cardiac Valve Formation in Developing Zebrafish
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- Development of the proepicardial organ in the zebrafish
- (2007) Fabrizio C. Serluca DEVELOPMENTAL BIOLOGY
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