A splicing variant of Merlin promotes metastasis in hepatocellular carcinoma
Published 2015 View Full Article
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Title
A splicing variant of Merlin promotes metastasis in hepatocellular carcinoma
Authors
Keywords
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Journal
Nature Communications
Volume 6, Issue 1, Pages -
Publisher
Springer Nature
Online
2015-10-07
DOI
10.1038/ncomms9457
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Related references
Note: Only part of the references are listed.- Merlin-Deficient Human Tumors Show Loss of Contact Inhibition and Activation of Wnt/β-Catenin Signaling Linked to the PDGFR/Src and Rac/PAK Pathways
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- Merlin, the NF2 Gene Product
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- NF2-deficient cells depend on the Rac1-canonical Wnt signaling pathway to promote the loss of contact inhibition of proliferation
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- Regulation of mixed lineage kinase 3 is required for Neurofibromatosis-2-mediated growth suppression in human cancer
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- Degradation of p21CDKN1A after DNA damage is independent of type of lesion, and is not required for DNA repair
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- A Global View of Cancer-Specific Transcript Variants by Subtractive Transcriptome-Wide Analysis
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- Merlin Is a Potent Inhibitor of Glioma Growth
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- Validation of the p21-Activated Kinases as Targets for Inhibition in Neurofibromatosis Type 2
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- Zeb1 links epithelial-mesenchymal transition and cellular senescence
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- Aberrant RNA splicing and its functional consequences in cancer cells
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- Identification of side population cells in human hepatocellular carcinoma cell lines with stepwise metastatic potentials
- (2008) Guo-Ming Shi et al. JOURNAL OF CANCER RESEARCH AND CLINICAL ONCOLOGY
- Functional inactivation of NF2/merlin in human mesothelioma
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- Merlin regulates transmembrane receptor accumulation and signaling at the plasma membrane in primary mouse Schwann cells and in human schwannomas
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- Magic but treatable? Tumours due to loss of Merlin
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