SAD-A kinase controls islet β-cell size and function as a mediator of mTORC1 signaling
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Title
SAD-A kinase controls islet β-cell size and function as a mediator of mTORC1 signaling
Authors
Keywords
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Journal
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Volume 110, Issue 34, Pages 13857-13862
Publisher
Proceedings of the National Academy of Sciences
Online
2013-08-07
DOI
10.1073/pnas.1307698110
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- mTORC1-Mediated Cell Proliferation, But Not Cell Growth, Controlled by the 4E-BPs
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- mTOR Inhibition by Rapamycin Prevents -Cell Adaptation to Hyperglycemia and Exacerbates the Metabolic State in Type 2 Diabetes
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- Tuberous sclerosis complex proteins control axon formation
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- Biphasic Response of Pancreatic -Cell Mass to Ablation of Tuberous Sclerosis Complex 2 in Mice
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- Disruption of Tsc2 in pancreatic cells induces cell mass expansion and improved glucose tolerance in a TORC1-dependent manner
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