Loss of p63 and its microRNA-205 target results in enhanced cell migration and metastasis in prostate cancer

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TAp63 suppresses metastasis through coordinate regulation of Dicer and miRNAs

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MicroRNA-205 inhibits tumor cell migration through down-regulating the expression of the LDL receptor-related protein 1

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miR-205 Exerts Tumor-Suppressive Functions in Human Prostate through Down-regulation of Protein Kinase C 

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Mutant p53 Drives Invasion by Promoting Integrin Recycling

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A Mutant-p53/Smad Complex Opposes p63 to Empower TGFβ-Induced Metastasis

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Insulin-like Growth Factor-I-Dependent Up-regulation of ZEB1 Drives Epithelial-to-Mesenchymal Transition in Human Prostate Cancer Cells

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Overexpression of ΔNp63 in a human nasopharyngeal carcinoma cell line downregulates CKIs and enhances cell proliferation

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The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

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