KLF17 empowers TGF-β/Smad signaling by targeting Smad3-dependent pathway to suppress tumor growth and metastasis during cancer progression
Published 2015 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
KLF17 empowers TGF-β/Smad signaling by targeting Smad3-dependent pathway to suppress tumor growth and metastasis during cancer progression
Authors
Keywords
-
Journal
Cell Death & Disease
Volume 6, Issue 3, Pages e1681
Publisher
Springer Nature
Online
2015-03-13
DOI
10.1038/cddis.2015.48
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- MicroRNA-1246 enhances migration and invasion through CADM1 in hepatocellular carcinoma
- (2014) Zhao Sun et al. BMC CANCER
- 14-3-3 σ is a new target up-regulated by transforming growth factor-β1 through a Smad3-dependent mechanism
- (2013) Hye-Young Hong et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Identification of KLF17 as a novel epithelial to mesenchymal transition inducer via direct activation of TWIST1 in endometrioid endometrial cancer
- (2013) Peixin Dong et al. CARCINOGENESIS
- AEG-1 participates in TGF-beta1-induced EMT through p38 MAPK activation
- (2013) Jiali Wei et al. CELL BIOLOGY INTERNATIONAL
- Epithelial stem cell mutations that promote squamous cell carcinoma metastasis
- (2013) Ruth A. White et al. JOURNAL OF CLINICAL INVESTIGATION
- Down-regulated KLF17 expression is associated with tumor invasion and poor prognosis in hepatocellular carcinoma
- (2013) Fu-Yao Liu et al. MEDICAL ONCOLOGY
- MicroRNA-9 enhances migration and invasion through KLF17 in hepatocellular carcinoma
- (2013) Zhao Sun et al. Molecular Oncology
- Differential regulation of the REGγ–proteasome pathway by p53/TGF-β signalling and mutant p53 in cancer cells
- (2013) Amjad Ali et al. Nature Communications
- TGF-β-Id1 Signaling Opposes Twist1 and Promotes Metastatic Colonization via a Mesenchymal-to-Epithelial Transition
- (2013) Marko Stankic et al. Cell Reports
- Transforming Growth Factor-β Directly Induces p53-up-regulated Modulator of Apoptosis (PUMA) during the Rapid Induction of Apoptosis in Myc-driven B-cell Lymphomas
- (2012) Lindsay C. Spender et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Global cancer statistics
- (2011) Ahmedin Jemal et al. CA-A CANCER JOURNAL FOR CLINICIANS
- Smad4 Inactivation Promotes Malignancy and Drug Resistance of Colon Cancer
- (2011) P. Papageorgis et al. CANCER RESEARCH
- Role of Smads in TGFβ signaling
- (2011) Carl-Henrik Heldin et al. CELL AND TISSUE RESEARCH
- Switching TGFβ from a tumor suppressor to a tumor promoter
- (2011) Gareth J Inman CURRENT OPINION IN GENETICS & DEVELOPMENT
- TGF-β signalling is mediated by two autonomously functioning TβRI:TβRII pairs
- (2011) Tao Huang et al. EMBO JOURNAL
- Estimates of worldwide burden of cancer in 2008: GLOBOCAN 2008
- (2010) Jacques Ferlay et al. INTERNATIONAL JOURNAL OF CANCER
- The dynamic roles of TGF-β in cancer
- (2010) Erik Meulmeester et al. JOURNAL OF PATHOLOGY
- Mechanism of TGF-β signaling to growth arrest, apoptosis, and epithelial–mesenchymal transition
- (2009) Carl-Henrik Heldin et al. CURRENT OPINION IN CELL BIOLOGY
- Antimetastatic Role of Smad4 Signaling in Colorectal Cancer
- (2009) Bixiang Zhang et al. GASTROENTEROLOGY
- KLF17 is a negative regulator of epithelial–mesenchymal transition and metastasis in breast cancer
- (2009) Kiranmai Gumireddy et al. NATURE CELL BIOLOGY
- KLF Family Members Regulate Intrinsic Axon Regeneration Ability
- (2009) D. L. Moore et al. SCIENCE
- Transcriptional regulation of metastatic [Id]entity by KLF17
- (2009) Marcin P Iwanicki et al. GENOME BIOLOGY
- TGFβ in Cancer
- (2008) Joan Massagué CELL
- A Very Private TGF-β Receptor Embrace
- (2008) Joan Massagué MOLECULAR CELL
Create your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create NowBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started