Mest but Not MiR-335 Affects Skeletal Muscle Growth and Regeneration

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miR-195/497 induce postnatal quiescence of skeletal muscle stem cells

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Mechanisms regulating skeletal muscle growth and atrophy

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MiR-335 Inhibits Small Cell Lung Cancer Bone Metastases via IGF-IR and RANKL Pathways

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The Imprinted H19 LncRNA Antagonizes Let-7 MicroRNAs

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Maternal imprinting at the H19–Igf2 locus maintains adult haematopoietic stem cell quiescence

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MicroRNA miR-335 is crucial for the BRCA1 regulatory cascade in breast cancer development

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miR-335 Directly Targets Rb1 (pRb/p105) in a Proximal Connection to p53-Dependent Stress Response

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miR-335 orchestrates cell proliferation, migration and differentiation in human mesenchymal stem cells

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H19 acts as a trans regulator of the imprinted gene network controlling growth in mice

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Pax3:Foxc2 Reciprocal Repression in the Somite Modulates Muscular versus Vascular Cell Fate Choice in Multipotent Progenitors

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An imprinted gene network that controls mammalian somatic growth is down-regulated during postnatal growth deceleration in multiple organs

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Disruption of the Interaction between Transcriptional Intermediary Factor 1β and Heterochromatin Protein 1 Leads to a Switch from DNA Hyper- to Hypomethylation and H3K9 to H3K27 Trimethylation on the MEST Promoter Correlating with Gene Reactivation

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Endogenous human microRNAs that suppress breast cancer metastasis

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