Inhibitory Phenotype of HBV-Specific CD4+ T-Cells Is Characterized by High PD-1 Expression but Absent Coregulation of Multiple Inhibitory Molecules
Published 2014 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Inhibitory Phenotype of HBV-Specific CD4+ T-Cells Is Characterized by High PD-1 Expression but Absent Coregulation of Multiple Inhibitory Molecules
Authors
Keywords
-
Journal
PLoS One
Volume 9, Issue 8, Pages e105703
Publisher
Public Library of Science (PLoS)
Online
2014-08-22
DOI
10.1371/journal.pone.0105703
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Broadly directed virus-specific CD4+T cell responses are primed during acute hepatitis C infection, but rapidly disappear from human blood with viral persistence
- (2012) Julian Schulze zur Wiesch et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Association of Differentiation State of CD4+ T Cells and Disease Progression in HIV-1 Perinatally Infected Children
- (2012) Elizabeth R. Sharp et al. PLoS One
- Upregulation of the Tim-3/Galectin-9 Pathway of T Cell Exhaustion in Chronic Hepatitis B Virus Infection
- (2012) Gaia Nebbia et al. PLoS One
- Progenitor and Terminal Subsets of CD8+ T Cells Cooperate to Contain Chronic Viral Infection
- (2012) M. A. Paley et al. SCIENCE
- Responsiveness of HIV-specific CD4 T cells to PD-1 blockade
- (2011) F. Porichis et al. BLOOD
- Surface expression patterns of negative regulatory molecules identify determinants of virus-specific CD8+ T-cell exhaustion in HIV infection
- (2011) T. Yamamoto et al. BLOOD
- Inhibitory Molecules That Regulate Expansion and Restoration of HCV-Specific CD4+ T Cells in Patients With Chronic Infection
- (2011) Bijan Raziorrouh et al. GASTROENTEROLOGY
- Role of the coinhibitory receptor cytotoxic T lymphocyte antigen-4 on apoptosis-Prone CD8 T cells in persistent hepatitis B virus infection
- (2011) Anna Schurich et al. HEPATOLOGY
- Human CD8+ and CD4+ T Cell Memory to Lymphocytic Choriomeningitis Virus Infection
- (2011) M. F. Kotturi et al. JOURNAL OF VIROLOGY
- Transcription factor T-bet represses expression of the inhibitory receptor PD-1 and sustains virus-specific CD8+ T cell responses during chronic infection
- (2011) Charlly Kao et al. NATURE IMMUNOLOGY
- The immunoregulatory role of CD244 in chronic hepatitis B infection and its inhibitory potential on virus-specific CD8+ T-cell function
- (2010) Bijan Raziorrouh et al. HEPATOLOGY
- Tim-3 expression on PD-1+ HCV-specific human CTLs is associated with viral persistence, and its blockade restores hepatocyte-directed in vitro cytotoxicity
- (2010) Rachel H. McMahan et al. JOURNAL OF CLINICAL INVESTIGATION
- Regulation of Virus-Specific CD4+ T Cell Function by Multiple Costimulatory Receptors during Chronic HIV Infection
- (2010) A. Kassu et al. JOURNAL OF IMMUNOLOGY
- Coexpression of PD-1, 2B4, CD160 and KLRG1 on Exhausted HCV-Specific CD8+ T Cells Is Linked to Antigen Recognition and T Cell Differentiation
- (2010) Bertram Bengsch et al. PLoS Pathogens
- Redefining Chronic Viral Infection
- (2009) Herbert W. Virgin et al. CELL
- Synergistic Reversal of Intrahepatic HCV-Specific CD8 T Cell Exhaustion by Combined PD-1/CTLA-4 Blockade
- (2009) Nobuhiro Nakamoto et al. PLoS Pathogens
- Functional Restoration of HCV-Specific CD8 T Cells by PD-1 Blockade Is Defined by PD-1 Expression and Compartmentalization
- (2008) Nobuhiro Nakamoto et al. GASTROENTEROLOGY
- Coregulation of CD8+ T cell exhaustion by multiple inhibitory receptors during chronic viral infection
- (2008) Shawn D Blackburn et al. NATURE IMMUNOLOGY
- Selective expansion of a subset of exhausted CD8 T cells by PD-L1 blockade
- (2008) S. D. Blackburn et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreFind the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
Search