Lack of Endogenous IL-10 Enhances Production of Proinflammatory Cytokines and Leads to Brucella abortus Clearance in Mice
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Title
Lack of Endogenous IL-10 Enhances Production of Proinflammatory Cytokines and Leads to Brucella abortus Clearance in Mice
Authors
Keywords
Brucella, Cytokines, Spleen, Granulomas, Immune response, T cells, Dendritic cells, Cloning
Journal
PLoS One
Volume 8, Issue 9, Pages e74729
Publisher
Public Library of Science (PLoS)
Online
2013-09-18
DOI
10.1371/journal.pone.0074729
References
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Note: Only part of the references are listed.- Central Role of MyD88-Dependent Dendritic Cell Maturation and Proinflammatory Cytokine Production to Control Brucella abortus Infection
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- CD4+ T Cell-derived IL-10 Promotes Brucella abortus Persistence via Modulation of Macrophage Function
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- Interleukin-17A genetic variants can confer resistance to brucellosis in Iranian population
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- Host Susceptibility toBrucella abortusInfection Is More Pronounced in IFN-γknockout than IL-12/β2-Microglobulin Double-Deficient Mice
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- (2011) Beata Clapp et al. INFECTION AND IMMUNITY
- B Cell–deficient Mice Display Markedly Enhanced Resistance to the Intracellular Bacterium Brucella abortus
- (2011) Radhika Goenka et al. JOURNAL OF INFECTIOUS DISEASES
- The Brucella abortus Phosphoglycerate Kinase Mutant Is Highly Attenuated and Induces Protection Superior to That of Vaccine Strain 19 in Immunocompromised and Immunocompetent Mice
- (2010) C. G. M. C. Trant et al. INFECTION AND IMMUNITY
- Regulatory T cells: how do they suppress immune responses?
- (2009) S. Sakaguchi et al. INTERNATIONAL IMMUNOLOGY
- Negative feedback control of the autoimmune response through antigen-induced differentiation of IL-10–secreting Th1 cells
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- Regulatory T cells exert checks and balances on self tolerance and autoimmunity
- (2009) Kajsa Wing et al. NATURE IMMUNOLOGY
- Pathological, Immunohistochemical and Bacteriological Study of Tissues and Milk of Cows and Fetuses Experimentally Infected with Brucella abortus
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