Synergistic Effect of Mesangial Cell-Induced CXCL1 and TGF-β1 in Promoting Podocyte Loss in IgA Nephropathy

The danger control concept in kidney disease: mesangial cells

(2013) Adriana Migliorini et al.   JOURNAL OF NEPHROLOGY

Relationships between Levels of Urinary Podocalyxin, Number of Urinary Podocytes, and Histologic Injury in Adult Patients with IgA Nephropathy

(2012) R. Asao et al.   Clinical Journal of the American Society of Nephrology

Chemokines in Renal Injury

(2011) A. C. K. Chung et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

IgA1 immune complexes from pediatric patients with IgA nephropathy activate cultured human mesangial cells

(2011) Jan Novak et al.   NEPHROLOGY DIALYSIS TRANSPLANTATION

Macromolecular IgA1 taken from patients with familial IgA Nephropathy or their asymptomatic relatives have higher reactivity to mesangial cells in vitro

(2009) Ka Ying Tam et al.   KIDNEY INTERNATIONAL

Aberrantly glycosylated IgA1 induces mesangial cells to produce platelet-activating factor that mediates nephrin loss in cultured podocytes

(2009) Rosanna Coppo et al.   KIDNEY INTERNATIONAL

Mechanical forces and TGF 1 reduce podocyte adhesion through  3 1 integrin downregulation

(2009) C. Dessapt et al.   NEPHROLOGY DIALYSIS TRANSPLANTATION